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Enhanced architecture and implementation of spectrum shaping codes.

ABSTRACT: Spectral shaping codes are modulation codes widely used in communication and data storage systems. This research enhances the algorithms employed in constructing spectral shaping codes for hardware implementation. We present a parallel scrambling calculation with a time complexity of O(1). Second, in the minimum accumulated signal power (MASP) module, the sine-cosine accumulation needs to be determined by remainder with time complexity O(n²). We offer reduced MASP computations for short bit-width data, ROM storage, and addition pipelines. It can remove the remainder operation, reducing accumulated complexity to O(1). In addition, we present a search algorithm to generate segmented lines to replace the square operations in the MASP module. By employing the search algorithm and shift operations, we can reduce the complexity of the square from O(n²) to O(1). The implementation results reveal that the original and proposed MASPs yield nearly identical spectrum nulls. The encoder-decoder of the spectral shaping codes with proposed approaches consumes just 6% of the hardware resources when carried out with a Spartan6 XC6SLX25.

SUBMITTER: Wang B

PROVIDER: S-EPMC10909226 | biostudies-literature | 2024

REPOSITORIES: biostudies-literature

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Enhanced architecture and implementation of spectrum shaping codes.

Wang Bingrui B Xie Zhaopeng Z Zhang Xingang X

PeerJ. Computer science 20240221

Spectral shaping codes are modulation codes widely used in communication and data storage systems. This research enhances the algorithms employed in constructing spectral shaping codes for hardware implementation. We present a parallel scrambling calculation with a time complexity of O(1). Second, in the minimum accumulated signal power (MASP) module, the sine-cosine accumulation needs to be determined by remainder with time complexity O(n2). We offer reduced MASP ...[more]

PMID: 38435626

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Project description:Behçet disease (BD) is a polygenic, multifactorial, multisystem inflammatory condition with unknown etiology. Global distribution of BD is geographically structured, highest prevalence observed among East Asian, Middle Eastern, and Mediterranean populations. Although adaptive selection on a few BD susceptibility loci is speculated, a thorough evolutionary analysis on the genetic architecture of BD is lacking. We aimed to understand whether increased BD risk in the human populations with high prevalence is due to past selection on BD associated genes. We performed population genetics analyses with East Asian (high BD prevalence), European (low/very low BD prevalence), and African (very low/no BD prevalence) populations. Comparison of ancestral and derived alleles' frequencies versus their reported susceptible or protective effect on BD showed both derived and ancestral alleles are associated with increased BD risk. Variants showing higher risk to and more significant association with BD had smaller allele frequency differences, and showed less population differentiation compared to variants that showed smaller risk and less significant association with BD. Results suggest BD alleles are not unique to East Asians but are also found in other world populations at appreciable frequencies, and argue against selection favoring these variants only in populations with high BD prevalence. BD associated gene analyses showed similar evolutionary histories driven by neutral processes for many genes or balancing selection for HLA (Human Leukocyte Antigen) genes in all three populations studied. However, nucleotide diversity in several HLA region genes was much higher in East Asians suggesting selection for high nucleotide and haplotype diversity in East Asians. Recent selective sweep for genes involved in antigen recognition, peptide processing, immune and cellular differentiation regulation was observed only in East Asians. We conclude that the evolutionary processes shaping the genetic diversity in BD risk genes are diverse, and elucidating the underlying specific selection mechanisms is complex. Several of the genes examined in this study are risk factors (such as ERAP1, IL23R, HLA-G) for other inflammatory diseases. Thus, our conclusions are not only limited to BD but may have broader implications for other inflammatory diseases.

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Enhanced architecture and implementation of spectrum shaping codes.

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Enhanced architecture and implementation of spectrum shaping codes.

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