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Molecular basis of the attenuation exhibited by molecularly cloned highly passaged chicken anemia virus isolates.


ABSTRACT: Chimeric virus experiments indicated that the pathogenicity and monoclonal antibody reactivity differences between two molecularly cloned, highly passaged chicken anemia virus isolates could be attributed to the VP1 amino acid change at residue 89. The introduction of this change into a pathogenic cloned low-passage isolate was not sufficient to cause attenuation.

SUBMITTER: Todd D 

PROVIDER: S-EPMC155136 | biostudies-literature | 2002 Aug

REPOSITORIES: biostudies-literature

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Molecular basis of the attenuation exhibited by molecularly cloned highly passaged chicken anemia virus isolates.

Todd Daniel D   Scott Alistair N J AN   Ball Neris W NW   Borghmans Borghert J BJ   Adair Brian M BM  

Journal of virology 20020801 16


Chimeric virus experiments indicated that the pathogenicity and monoclonal antibody reactivity differences between two molecularly cloned, highly passaged chicken anemia virus isolates could be attributed to the VP1 amino acid change at residue 89. The introduction of this change into a pathogenic cloned low-passage isolate was not sufficient to cause attenuation. ...[more]

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