Ontology highlight
ABSTRACT:
SUBMITTER: Bailey-Wilson JE
PROVIDER: S-EPMC3495053 | biostudies-literature | 2012 Jun
REPOSITORIES: biostudies-literature
Bailey-Wilson Joan E JE Childs Erica J EJ Cropp Cheryl D CD Schaid Daniel J DJ Xu Jianfeng J Camp Nicola J NJ Cannon-Albright Lisa A LA Farnham James M JM George Asha A Powell Isaac I Carpten John D JD Giles Graham G GG Hopper John L JL Severi Gianluca G English Dallas R DR Foulkes William D WD Mæhle Lovise L Møller Pål P Eeles Rosalind R Easton Douglas D Guy Michelle M Edwards Steve S Badzioch Michael D MD Whittemore Alice S AS Oakley-Girvan Ingrid I Hsieh Chih-Lin CL Dimitrov Latchezar L Stanford Janet L JL Karyadi Danielle M DM Deutsch Kerry K McIntosh Laura L Ostrander Elaine A EA Wiley Kathleen E KE Isaacs Sarah D SD Walsh Patrick C PC Thibodeau Stephen N SN McDonnell Shannon K SK Hebbring Scott S Lange Ethan M EM Cooney Kathleen A KA Tammela Teuvo L J TL Schleutker Johanna J Maier Christiane C Bochum Sylvia S Hoegel Josef J Grönberg Henrik H Wiklund Fredrik F Emanuelsson Monica M Cancel-Tassin Geraldine G Valeri Antoine A Cussenot Olivier O Isaacs William B WB
BMC medical genetics 20120619
<h4>Background</h4>Genetic variants are likely to contribute to a portion of prostate cancer risk. Full elucidation of the genetic etiology of prostate cancer is difficult because of incomplete penetrance and genetic and phenotypic heterogeneity. Current evidence suggests that genetic linkage to prostate cancer has been found on several chromosomes including the X; however, identification of causative genes has been elusive.<h4>Methods</h4>Parametric and non-parametric linkage analyses were perf ...[more]