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The social contagion of generosity.

ABSTRACT: Why do people help strangers when there is a low probability that help will be directly reciprocated or socially rewarded? A possible explanation is that these acts are contagious: those who receive or observe help from a stranger become more likely to help others. We test two mechanisms for the social contagion of generosity among strangers: generalized reciprocity (a recipient of generosity is more likely to pay it forward) and third-party influence (an observer of generous behavior is more likely to emulate it). We use an online experiment with randomized trials to test the two hypothesized mechanisms and their interaction by manipulating the extent to which participants receive and observe help. Results show that receiving help can increase the willingness to be generous towards others, but observing help can have the opposite effect, especially among those who have not received help. These results suggest that observing widespread generosity may attenuate the belief that one's own efforts are needed.

SUBMITTER: Tsvetkova M

PROVIDER: S-EPMC3923723 | biostudies-literature | 2014

REPOSITORIES: biostudies-literature

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The social contagion of generosity.

Tsvetkova Milena M Macy Michael W MW

PloS one 20140213 2

Why do people help strangers when there is a low probability that help will be directly reciprocated or socially rewarded? A possible explanation is that these acts are contagious: those who receive or observe help from a stranger become more likely to help others. We test two mechanisms for the social contagion of generosity among strangers: generalized reciprocity (a recipient of generosity is more likely to pay it forward) and third-party influence (an observer of generous behavior is more li ...[more]

PMID: 24551053

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Project description:Many behavioral phenomena have been found to spread interpersonally through social networks, in a manner similar to infectious diseases. An important difference between social contagion and traditional infectious diseases, however, is that behavioral phenomena can be acquired by non-social mechanisms as well as through social transmission. We introduce a novel theoretical framework for studying these phenomena (the SISa model) by adapting a classic disease model to include the possibility for 'automatic' (or 'spontaneous') non-social infection. We provide an example of the use of this framework by examining the spread of obesity in the Framingham Heart Study Network. The interaction assumptions of the model are validated using longitudinal network transmission data. We find that the current rate of becoming obese is 2 per year and increases by 0.5 percentage points for each obese social contact. The rate of recovering from obesity is 4 per year, and does not depend on the number of non-obese contacts. The model predicts a long-term obesity prevalence of approximately 42, and can be used to evaluate the effect of different interventions on steady-state obesity. Model predictions quantitatively reproduce the actual historical time course for the prevalence of obesity. We find that since the 1970s, the rate of recovery from obesity has remained relatively constant, while the rates of both spontaneous infection and transmission have steadily increased over time. This suggests that the obesity epidemic may be driven by increasing rates of becoming obese, both spontaneously and transmissively, rather than by decreasing rates of losing weight. A key feature of the SISa model is its ability to characterize the relative importance of social transmission by quantitatively comparing rates of spontaneous versus contagious infection. It provides a theoretical framework for studying the interpersonal spread of any state that may also arise spontaneously, such as emotions, behaviors, health states, ideas or diseases with reservoirs.

Dataset Information

The social contagion of generosity.

Publications

The social contagion of generosity.

Similar Datasets

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