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Microvascular Vasodilator Plasticity After Acute Exercise.


ABSTRACT: Endothelium-dependent vasodilation is reduced after acute exercise or after high intraluminal pressure in isolated arterioles from sedentary adults but not in arterioles from regular exercisers. The preserved vasodilation in arterioles from exercisers is hydrogen peroxide (H2O2) dependent, whereas resting dilation is nitric oxide (NO) dependent. We hypothesize chronic exercise elicits adaptations allowing for maintained vasodilation when NO bioavailability is reduced.

SUBMITTER: Robinson AT 

PROVIDER: S-EPMC5735031 | biostudies-literature | 2018 Jan

REPOSITORIES: biostudies-literature

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Microvascular Vasodilator Plasticity After Acute Exercise.

Robinson Austin T AT   Fancher Ibra S IS   Mahmoud Abeer M AM   Phillips Shane A SA  

Exercise and sport sciences reviews 20180101 1


Endothelium-dependent vasodilation is reduced after acute exercise or after high intraluminal pressure in isolated arterioles from sedentary adults but not in arterioles from regular exercisers. The preserved vasodilation in arterioles from exercisers is hydrogen peroxide (H2O2) dependent, whereas resting dilation is nitric oxide (NO) dependent. We hypothesize chronic exercise elicits adaptations allowing for maintained vasodilation when NO bioavailability is reduced. ...[more]

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