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A short certificateless aggregate signature against coalition attacks.

ABSTRACT: Certificateless aggregate signature (CLAS) is a crucial cryptosystem. It can not only compress multiple signatures into a short signature, but also ensure the validity of each signature participating in the aggregation by verifying the validity of an resulting aggregate signature. Therefore, a secure and efficient CLAS scheme is very useful for resource-constrained environments because it greatly reduces the overall length of the signature and the verifier's computational overhead. Cheng et al. presented an efficient CLAS scheme and proved its security in the random oracle model. However, we find that their scheme has security flaws. In this paper, we demonstrate that Cheng et al.'s CLAS scheme is vulnerable to coalition attacks from internal signers. To overcome these attacks, we present an improved CLAS scheme and prove that it is existentially unforgeable under the computational Diffie-Hellman assumption. In addition, our CLAS scheme can not only resist coalition attacks but also generate a very short aggregate signature. The performance analysis results show that our improved CLAS scheme is lower than the related CLAS schemes in terms of communication overhead and computation cost.

SUBMITTER: Yang X

PROVIDER: S-EPMC6291108 | biostudies-literature | 2018

REPOSITORIES: biostudies-literature

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A short certificateless aggregate signature against coalition attacks.

Yang Xiaodong X Wang Jinli J Ma Tingchun T Li Yutong Y Wang Caifen C

PloS one 20181212 12

Certificateless aggregate signature (CLAS) is a crucial cryptosystem. It can not only compress multiple signatures into a short signature, but also ensure the validity of each signature participating in the aggregation by verifying the validity of an resulting aggregate signature. Therefore, a secure and efficient CLAS scheme is very useful for resource-constrained environments because it greatly reduces the overall length of the signature and the verifier's computational overhead. Cheng et al. ...[more]

PMID: 30540746

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Project description:Bacterial competition may rely on secretion systems such as the type 6 secretion system (T6SS), which punctures and releases toxic molecules into neighboring cells. To subsist, bacterial targets must counteract the threats posed by T6SS-positive competitors. In this study, we used a comprehensive genome-wide high-throughput screening approach to investigate the dynamics of interbacterial competition. Our primary goal was to identify deletion mutants within the well-characterized E. coli K-12 single-gene deletion library, the Keio collection, that demonstrated resistance to T6SS-mediated killing by the enteropathogenic bacterium Cronobacter malonaticus. We identified 49 potential mutants conferring resistance to T6SS and focused our interest on a deletion mutant (∆fimE) exhibiting enhanced expression of type 1 fimbriae. We demonstrated that the presence of type 1 fimbriae leads to the formation of microcolonies and thus protects against T6SS-mediated assaults. Collectively, our study demonstrated that adhesive structures such as type 1 fimbriae confer collective protective behavior against T6SS attacks.IMPORTANCEType 6 secretion systems (T6SS) are molecular weapons employed by gram-negative bacteria to eliminate neighboring microbes. T6SS plays a pivotal role as a virulence factor, enabling pathogenic gram-negative bacteria to compete with the established communities to colonize hosts and induce infections. Gaining a deeper understanding of bacterial interactions will allow the development of strategies to control the action of systems such as the T6SS that can manipulate bacterial communities. In this context, we demonstrate that bacteria targeted by T6SS attacks from the enteric pathogen Cronobacter malonaticus, which poses a significant threat to infants, can develop a collective protective mechanism centered on the production of type I fimbriae. These adhesive structures promote the aggregation of bacterial preys and the formation of microcolonies, which protect the cells from T6SS attacks.

Dataset Information

A short certificateless aggregate signature against coalition attacks.

Publications

A short certificateless aggregate signature against coalition attacks.

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