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Effect of deforestation on access to clean drinking water.

ABSTRACT: Using satellite data on deforestation and weather in Malawi and linking those datasets with household survey datasets, we estimate the causal effect of deforestation on access to clean drinking water. In the existing literature on forest science and hydrology, the consensus is that deforestation increases water yield. In this study, we directly examine the causal effect of deforestation on households' access to clean drinking water. Results of the two-stage least-squares (2SLS) with cluster and time fixed-effect estimations illustrate strong empirical evidence that deforestation decreases access to clean drinking water. Falsification tests show that the possibility of our instrumental variable picking up an unobserved time trend is very unlikely. We find that a 1.0-percentage-point increase in deforestation decreases access to clean drinking water by 0.93 percentage points. With this estimated impact, deforestation in the last decade in Malawi (14%) has had the same magnitude of effect on access to clean drinking water as that of a 9% decrease in rainfall.

SUBMITTER: Mapulanga AM

PROVIDER: S-EPMC6486726 | biostudies-literature | 2019 Apr

REPOSITORIES: biostudies-literature

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Effect of deforestation on access to clean drinking water.

Mapulanga Annie Mwayi AM Naito Hisahiro H

Proceedings of the National Academy of Sciences of the United States of America 20190325 17

Using satellite data on deforestation and weather in Malawi and linking those datasets with household survey datasets, we estimate the causal effect of deforestation on access to clean drinking water. In the existing literature on forest science and hydrology, the consensus is that deforestation increases water yield. In this study, we directly examine the causal effect of deforestation on households' access to clean drinking water. Results of the two-stage least-squares (2SLS) with cluster and ...[more]

PMID: 30910966

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Project description:Concentration- and time-dependent genomic changes in the mouse urinary bladder following exposure to arsenate in drinking water for up to twelve weeks. Inorganic arsenic (Asi) is a known human bladder carcinogen. The objective of this study was to examine the concentration dependence of the genomic response to Asi in the urinary bladders of mice. C57BL/6J mice were exposed for 1 or 12 weeks to arsenate in drinking water at concentrations of 0.5, 2, 10, and 50 mg As/L. Urinary bladders were analyzed using gene expression microarrays. A consistent reversal was observed in the direction of gene expression change: from predominantly decreased expression at 1 week to predominantly increased expression at 12 weeks. These results are consistent with evidence from in vitro studies of an acute adaptive response that is suppressed on longer exposure due to down-regulation of Fos. Pathways with the highest enrichment in gene expression changes were associated with epithelial-to-mesenchymal transition, inflammation, and proliferation. Benchmark dose (BMD) analysis determined that the lowest median BMD values for pathways were above 5 mg As/L, despite the fact that pathway enrichment was observed at the 0.5 mg As/L exposure concentration. This disparity may result from the non-monotonic nature of the concentration-responses for the expression changes of a number of genes, as evidenced by the much fewer gene expression changes at 2 mg As/L compared to lower or higher concentrations. Pathway categories with concentration-related gene expression changes included cellular morphogenesis, inflammation, apoptosis/survival, cell cycle control, and DNA damage response. The results of this study provide evidence of a concentration-dependent transition in the mode of action for the subchronic effects of Asi in mouse bladder cells in the vicinity of 2 mg Asi/L. Female C57Bl/J mice will be exposed to COLD Arsenate in drinking water. One week interium sac on 7/18/06. 100 samples including liver, lung, kidney and bladder. Bladder will be analyzed with microarrays, 24 samples. Twelve week terminal sac on 10/05/06. 75 total samples including lung, kidney, and bladder. Bladdler will be analyzed by microarray, 25 samples. Drinking water containing As will be prepared weekly with monitoring to determine amount used by mice. Following tissues will be available for genomic study: Bladder, liver, lung, and kidney.

Dataset Information

Effect of deforestation on access to clean drinking water.

Publications

Effect of deforestation on access to clean drinking water.

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