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The required number of trials for biomechanical analysis of a golf swing.

ABSTRACT: The increasing interest in the biomechanical analysis of the golf swing warrants establishing the minimum number of trials required to obtain reliable data. Several such methods have been suggested previously for other movement tasks, and it has been shown that the number of required trials depends on the method used and on the task examined. This study aimed to compare three methods of reliability: a sequential average, intraclass correlations, and a modified version of the standard error of measurement (SEMind). Kinematic and kinetic data of 10 recreational golfers performing 15 shots with both a six-iron and a driver was collected using a ten-camera motion capture system and force platforms. Range-of-motion, velocity, joint moments, and ground reaction forces were extracted and analysed using the three methods. The sequential average method yielded the highest number of required trials (12), while the intraclass correlations and SEMind both resulted in lower numbers of required trials (4). Considering the variability between participants and strengths and limitations of the various methods, we conclude that 8 trials is sufficient for biomechanical analyses of a golf swing and recommend the SEMind method for determining how many swings should be collected.

SUBMITTER: Severin AC

PROVIDER: S-EPMC6706322 | biostudies-literature | 2019 Jan

REPOSITORIES: biostudies-literature

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The required number of trials for biomechanical analysis of a golf swing.

Severin Anna C AC Barnes Sally G SG Tackett Stewart A SA Barnes C Lowry CL Mannen Erin M EM

Sports biomechanics 20190130 2

The increasing interest in the biomechanical analysis of the golf swing warrants establishing the minimum number of trials required to obtain reliable data. Several such methods have been suggested previously for other movement tasks, and it has been shown that the number of required trials depends on the method used and on the task examined. This study aimed to compare three methods of reliability: a sequential average, intraclass correlations, and a modified version of the standard error of me ...[more]

PMID: 30698499

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Project description:BackgroundSuccessful walking requires the execution of the pre-swing biomechanical tasks of body propulsion and leg swing initiation, which are often impaired post-stroke. While excess rectus femoris activity during swing is often associated with low knee flexion, previous work has suggested that deficits in propulsion and leg swing initiation may also contribute. The purpose of this study was to determine underlying causes of propulsion, leg swing initiation and knee flexion deficits in pre-swing and their link to stiff knee gait in individuals post-stroke.MethodsMusculoskeletal models and forward dynamic simulations were developed for individuals post-stroke (n = 15) and healthy participants (n = 5). Linear regressions were used to evaluate the relationships between peak knee flexion, braking and propulsion symmetry, and individual muscle contributions to braking, propulsion, knee flexion in pre-swing, and leg swing initiation.ResultsFour out of fifteen of individuals post-stroke had higher plantarflexor contributions to propulsion and seven out of fifteen had higher vasti contributions to braking on their paretic leg relative to their nonparetic leg. Higher gastrocnemius contributions to propulsion predicted paretic propulsion symmetry (p = 0.005) while soleus contributions did not. Higher vasti contributions to braking in pre-swing predicted lower knee flexion (p = 0.022). The rectus femoris had minimal contributions to lower knee flexion acceleration in pre-swing compared to contributions from the vasti. However, for some individuals with low knee flexion, during pre-swing the rectus femoris absorbed more power and the iliopsoas contributed less power to the paretic leg. Total musculotendon work done on the paretic leg in pre-swing did not predict knee flexion during swing.ConclusionsThese results emphasize the multiple causes of propulsion asymmetry in individuals post-stroke, including low plantarflexor contributions to propulsion, increased vasti contributions to braking and reliance on compensatory mechanisms. The results also show that the rectus femoris is not a major contributor to knee flexion in pre-swing, but absorbs more power from the paretic leg in pre-swing in some individuals with stiff knee gait. These results highlight the need to identify individual causes of propulsion and knee flexion deficits to design more effective rehabilitation strategies.

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The required number of trials for biomechanical analysis of a golf swing.

Publications

The required number of trials for biomechanical analysis of a golf swing.

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