Dataset Information

Can Milk Affect Recovery from Simulated Team-Sport Match Play?

ABSTRACT: This study investigated the effects of cow's milk on recovery from repeated simulated team games (STGs) in females. Twenty female team-sport athletes completed an STG circuit (2x ~ 30 min, with 10 min 'half-time'). Measures of muscle function, soreness and tiredness, symptoms of stress and serum markers of muscle damage and oxidative stress were determined pre- and 24 h, 48 h, 72 h and 96 h following the circuit. At 48 h, a second STG was completed. Sprint performance (5 m, 15 m), lap time, countermovement jump (CMJ), heart rate and RPE were recorded during each STG. Immediately following each STG, participants consumed either 500 mL of cow's milk (MILK) or 500 mL of an energy-matched carbohydrate (CHO) solution. Compared to CHO, MILK had a beneficial effect in attenuating losses in peak torque for knee extension (60°/s) (likely; effect size (ES) = 0.26 to 0.28) knee flexion (60°/s) (likely; ES = 0.45 to 0.61). A benefit for MILK was observed for 5 m sprint (possible-likely; ES = 0.40 to 0.58), 10 m sprint (likely; ES = 0.30 to 0.53) and symptoms of stress (likely-very likely, small). Mostly unclear outcomes for other variables were observed. For STG variables, trivial (HR, CMJ) and unclear (5 m sprint, 15 m sprint, lap-time, RPE) outcomes were recorded. In conclusion, the consumption of 500 mL of milk attenuated losses in muscle function and perceptions of stress following repeated simulated team-sports games. However, further investigation is warranted to determine whether MILK can influence subsequent team-sport performance.

SUBMITTER: Rankin P

PROVIDER: S-EPMC7019310 | biostudies-literature | 2019 Dec

REPOSITORIES: biostudies-literature

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Can Milk Affect Recovery from Simulated Team-Sport Match Play?

Rankin Paula P Callanan Danielle D O'Brien Kevin K Davison Gareth G Stevenson Emma J EJ Cockburn Emma E

Nutrients 20191231 1

This study investigated the effects of cow's milk on recovery from repeated simulated team games (STGs) in females. Twenty female team-sport athletes completed an STG circuit (2x ~ 30 min, with 10 min 'half-time'). Measures of muscle function, soreness and tiredness, symptoms of stress and serum markers of muscle damage and oxidative stress were determined pre- and 24 h, 48 h, 72 h and 96 h following the circuit. At 48 h, a second STG was completed. Sprint performance (5 m, 15 m), lap time, coun ...[more]

PMID: 31906239

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Project description:The aim of the current investigation was to identify the effects of scheduled carbohydrate (CHO) and caffeine (CAF) supplementation on simulated team sport match-play performance. Ten male hurling players completed three hurling match-play simulation protocols (HSP) performed 7 days apart in a double-blind, randomized design. Supplementation included CHO, CHO + CAF, and placebo (PLA). In a randomized order, participants ingested either a 6% CHO solution, a PLA solution of similar taste, or a combined intake of 6% CHO solution + 200 mg CAF capsule. At specific time points (Pre-0 min; half time (HT)-30 min; full time (FT)-60 min), participants completed a repeated sprint protocol (RAST; 12 × 20 m). Physiological [% maximal oxygen uptake (%VO2max), % mean oxygen uptake (%VO2mean), % maximal heart rate (%HRmax), % mean heart rate (%HRmean), respiratory exchange ratio (RER), and blood lactate (BLa)] and performance [(best sprint time (RSAbest), mean sprint time (RSAmean), and rate of perceived exertion (RPE)] variables were monitored throughout each simulation. Non-significant differences were observed between supplement trials (CHO, CHO + CAF, and PLA) for BLa (?2 = 0.001, small), %VO2max (?2 = 0.001, small), %VO2mean (?2 = 0.004, small), %HRmax (?2 = 0.007, small), %HRmean (?2 = 0.018, small), RER (?2 = 0.007, small), RPE (?2 = 0.007, small), and RSAbest (?2 = 0.050, small). RSAmean performance significantly improved in CHO + CAF trials compared to PLA, with sprint times significantly improved from Pre to FT also (?2 = 0.135, medium). A significant difference was observed in BLa between time points (Pre, HT, and FT) (?2 = 0.884, large) in % HRmax (?2 = 0.202, medium), %HRmean (?2 = 0.477, large), and RER (?2 = 0.554, large) across halves and in RPE across time points (?2 = 0.670, large). Our data provide novel data regarding the effects of CHO and CAF supplementation on team sport performance, with co-ingestion of CHO + CAF reducing the decrement in repeated sprint performance compared to PLA.

Project description:Aim: Previous research into the etiology of neuromuscular fatigue following competitive soccer match-play has primarily focused on peripheral perturbations, with limited research assessing central nervous system function in the days post-match. The aim of the present study was to examine the contribution and time-course of recovery of central and peripheral factors toward neuromuscular fatigue following competitive soccer match-play. Methods: Sixteen male semi-professional soccer players completed a 90-min soccer match. Pre-, post- and at 24, 48, and 72 h participants completed a battery of neuromuscular, physical, and perceptual tests. Maximal voluntary contraction force (MVC) and twitch responses to electrical (femoral nerve) and transcranial magnetic stimulation (TMS) of the motor cortex during isometric knee-extension and at rest were measured to assess central nervous system (voluntary activation, VA) and muscle contractile (potentiated twitch force, Qtw, pot) function. Electromyography responses of the rectus femoris to single- and paired-pulse TMS were used to assess corticospinal excitability and short-interval intracortical inhibition (SICI), respectively. Fatigue and perceptions of muscle soreness were assessed via visual analog scales, and physical function was assessed through measures of jump (countermovement jump height and reactive strength index) and sprint performance. Results: Competitive match-play elicited significant post-match declines in MVC force (-14%, P < 0.001) that persisted for 48 h (-4%, P = 0.01), before recovering by 72 h post-exercise. VA (motor point stimulation) was reduced immediately post-match (-8%, P < 0.001), and remained depressed at 24 h (-5%, P = 0.01) before recovering by 48 h post-exercise. Qtw,pot was reduced post-match (-14%, P < 0.001), remained depressed at 24 h (-6%, P = 0.01), before recovering by 48 h post-exercise. No changes were evident in corticospinal excitability or SICI. Jump performance took 48 h to recover, while perceptions of fatigue persisted at 72 h. Conclusion: Competitive soccer match-play elicits substantial impairments in central nervous system and muscle function, requiring up to 48 h to resolve. The results of the study could have important implications for fixture scheduling, the optimal management of the training process, squad rotation during congested competitive schedules, and the implementation of appropriate recovery interventions.

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Project description:Our study aimed to investigate changes of different markers for routine assessment of fatigue and recovery in response to high-intensity interval training (HIIT).22 well-trained male and female team sport athletes (age, 23.0 ± 2.7 years; V?O2 max, 57.6 ± 8.6 mL · min · kg(-1)) participated in a six-day running-based HIIT-microcycle with a total of eleven HIIT sessions. Repeated sprint ability (RSA; criterion measure of fatigue and recovery), countermovement jump (CMJ) height, jump efficiency in a multiple rebound jump test (MRJ), 20-m sprint performance, muscle contractile properties, serum concentrations of creatinkinase (CK), c-reactive protein (CRP) and urea as well as perceived muscle soreness (DOMS) were measured pre and post the training program as well as after 72 h of recovery.Following the microcycle significant changes (p < 0.05) in RSA as well as in CMJ and MRJ performance could be observed, showing a decline (%? ± 90% confidence limits, ES = effect size; RSA: -3.8 ± 1.0, ES = -1.51; CMJ: 8.4 ± 2.9, ES = -1.35; MRJ: 17.4 ± 4.5, ES = -1.60) and a return to baseline level (RSA: 2.8 ± 2.6, ES = 0.53; CMJ: 4.1 ± 2.9, ES = 0.68; MRJ: 6.5 ± 4.5, ES = 0.63) after 72 h of recovery. Athletes also demonstrated significant changes (p < 0.05) in muscle contractile properties, CK, and DOMS following the training program and after the recovery period. In contrast, CRP and urea remained unchanged throughout the study. Further analysis revealed that the accuracy of markers for assessment of fatigue and recovery in comparison to RSA derived from a contingency table was insufficient. Multiple regression analysis also showed no correlations between changes in RSA and any of the markers.Mean changes in measures of neuromuscular function, CK and DOMS are related to HIIT induced fatigue and subsequent recovery. However, low accuracy of a single or combined use of these markers requires the verification of their applicability on an individual basis.

Project description:Collective team collapse occurs when multiple players of a sport team experience a sudden and extreme underperformance within a game. To date, minimal research has been conducted on the causes of collective team collapse. Thus, goals of this study were to explore perceived causes of collective team collapse in different sports and to define team collapse in contrast to negative momentum. To investigate factors causing and maintaining collective sport team collapse, an inductive, exploratory qualitative analysis of individual interviews was conducted. Semi-structured interviews were carried out with 10 athletes of professional German teams of various sports playing in between first and fourth division. Participants were interviewed about a team collapse event they had experienced with their team during the past year. Data were collected and analyzed using a grounded theory methodology. Collective team collapse appeared to be induced by a temporal cascade of causes rather than by single triggers. This cascade included antecedents, which represent factors that make the occurrence of a team collapse more likely; critical events, which include specific events within the game that trigger a team collapse; as well as affective, cognitive, and behavioral outcomes that foster a maintenance of the collapse. Within this theoretical framework, social factors, such as decreased performance contagion or emotional contagion, played crucial roles in causing a team collapse. These results illustrate that collective team collapse is more than the sum of individual choking of multiple players at the same time. In conclusion, a new definition, differentiating team collapse from negative momentum, is introduced. Furthermore, a process model of causes of collective team collapse is proposed. The results provide first insights into causes of collective collapse in a variety of team sports. The developed model is supposed to help future research to better connect to practice and to support athletes, coaches, and sport psychologists.

Dataset Information

Can Milk Affect Recovery from Simulated Team-Sport Match Play?

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Can Milk Affect Recovery from Simulated Team-Sport Match Play?

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