Ontology highlight
ABSTRACT:
SUBMITTER: Bikdeli B
PROVIDER: S-EPMC7164881 | biostudies-literature | 2020 Jun
REPOSITORIES: biostudies-literature
Bikdeli Behnood B Madhavan Mahesh V MV Jimenez David D Chuich Taylor T Dreyfus Isaac I Driggin Elissa E Nigoghossian Caroline Der C Ageno Walter W Madjid Mohammad M Guo Yutao Y Tang Liang V LV Hu Yu Y Giri Jay J Cushman Mary M Quéré Isabelle I Dimakakos Evangelos P EP Gibson C Michael CM Lippi Giuseppe G Favaloro Emmanuel J EJ Fareed Jawed J Caprini Joseph A JA Tafur Alfonso J AJ Burton John R JR Francese Dominic P DP Wang Elizabeth Y EY Falanga Anna A McLintock Claire C Hunt Beverley J BJ Spyropoulos Alex C AC Barnes Geoffrey D GD Eikelboom John W JW Weinberg Ido I Schulman Sam S Carrier Marc M Piazza Gregory G Beckman Joshua A JA Steg P Gabriel PG Stone Gregg W GW Rosenkranz Stephan S Goldhaber Samuel Z SZ Parikh Sahil A SA Monreal Manuel M Krumholz Harlan M HM Konstantinides Stavros V SV Weitz Jeffrey I JI Lip Gregory Y H GYH
Journal of the American College of Cardiology 20200417 23
Coronavirus disease-2019 (COVID-19), a viral respiratory illness caused by the severe acute respiratory syndrome-coronavirus-2 (SARS-CoV-2), may predispose patients to thrombotic disease, both in the venous and arterial circulations, because of excessive inflammation, platelet activation, endothelial dysfunction, and stasis. In addition, many patients receiving antithrombotic therapy for thrombotic disease may develop COVID-19, which can have implications for choice, dosing, and laboratory monit ...[more]