Dataset Information

Rapid Low-Dimensional Li+ Ion Hopping Processes in Synthetic Hectorite-Type Li0.5[Mg2.5Li0.5]Si4O10F2.

ABSTRACT: Understanding the origins of fast ion transport in solids is important to develop new ionic conductors for batteries and sensors. Nature offers a rich assortment of rather inspiring structures to elucidate these origins. In particular, layer-structured materials are prone to show facile Li+ transport along their inner surfaces. Here, synthetic hectorite-type Li0.5[Mg2.5Li0.5]Si4O10F2, being a phyllosilicate, served as a model substance to investigate Li+ translational ion dynamics by both broadband conductivity spectroscopy and diffusion-induced 7Li nuclear magnetic resonance (NMR) spin-lattice relaxation experiments. It turned out that conductivity spectroscopy, electric modulus data, and NMR are indeed able to detect a rapid 2D Li+ exchange process governed by an activation energy as low as 0.35 eV. At room temperature, the bulk conductivity turned out to be in the order of 0.1 mS cm-1. Thus, the silicate represents a promising starting point for further improvements by crystal chemical engineering. To the best of our knowledge, such a high Li+ ionic conductivity has not been observed for any silicate yet.

SUBMITTER: Hiebl C

PROVIDER: S-EPMC7499405 | biostudies-literature | 2020 Sep

REPOSITORIES: biostudies-literature

ACCESS DATA

Publications

Rapid Low-Dimensional Li+ Ion Hopping Processes in Synthetic Hectorite-Type Li0.5[Mg2.5Li0.5]Si4O10F2.

Hiebl Caroline C Loch Patrick P Brinek Marina M Gombotz Maria M Gadermaier Bernhard B Heitjans Paul P Breu Josef J Wilkening H Martin R HMR

Chemistry of materials : a publication of the American Chemical Society 20200805 17

Understanding the origins of fast ion transport in solids is important to develop new ionic conductors for batteries and sensors. Nature offers a rich assortment of rather inspiring structures to elucidate these origins. In particular, layer-structured materials are prone to show facile Li+ transport along their inner surfaces. Here, synthetic hectorite-type Li0.5[Mg2.5Li0.5]Si4O10F2, being a phyllosilicate, served as a ...[more]

PMID: 32952297

Similar Datasets

Project description:Previous studies have shown that long-term exposure to fine particulate matter (PM2.5) increases the morbidity and mortality of pulmonary diseases such as asthma, chronic obstructive pulmonary disease and pulmonary emphysema. Oxidative stress and inflammation play key roles in pulmonary damage caused by PM2.5. Nuclear factor erythroid 2-related factor 2 (Nrf2) could regulate the expression of antioxidant and anti-inflammatory genes and is pivotal for protection against PM2.5-induced oxidative stress. In this study, a real-ambient exposure system was constructed with the outdoor ambient air in north China. Wild-type (WT) and Nrf2-/- (KO) mice were exposed to the real-ambient system for six weeks. After PM2.5 exposure, our data showed that the levels of inflammatory factors and malondialdehyde were significantly increased in WT and KO mice. Moreover, the lung function and pathological phenotype of the WT mice were altered but there was no obvious change in the Nrf2-/- mice. To further explore the potential molecular mechanisms, we performed RNA-sequencing. The RNA-sequence analysis results showed that the CYP450 pathway in the first ten pathways of KEGG was related to the metabolism of PM2.5. In WT and KO mice, the expression of CYP2E1 in the CYP450 pathway showed opposite trends after PM2.5 exposure. The data showed that the expression of the CYP2E1 gene in WT-PM mice increased while it decreased in KO-PM; the expression of the CYP2E1 protein showed a similar trend. CYP2E1 is primarily distributed in the endoplasmic reticulum (ER) where it could metabolize various exogenous substances attached to PM2.5 and produce highly toxic oxidation products closely related to ER stress. Consistently, the expression level of GRP94, a biomarker of ER stress, was increased in WT mice and reduced in KO mice under PM2.5 exposure. Persistent ER stress is a mechanism that causes lung damage under PM2.5 exposure. Nrf2 facilitates lung injury during PM2.5 exposure and CYP2E1 metabolism is involved in this process.

Dataset Information

Rapid Low-Dimensional Li+ Ion Hopping Processes in Synthetic Hectorite-Type Li0.5[Mg2.5Li0.5]Si4O10F2.

Publications

Rapid Low-Dimensional Li<sup>+</sup> Ion Hopping Processes in Synthetic Hectorite-Type Li<sub>0.5</sub>[Mg<sub>2.5</sub>Li<sub>0.5</sub>]Si<sub>4</sub>O<sub>10</sub>F<sub>2</sub>.

Similar Datasets

OmicsDI is part of the ELIXIR infrastructure