Dataset Information

Occupational Exposure during Asphalt Paving-Comparison of Hot and Warm Mix Asphalt in Field Experiments.

ABSTRACT:

Objectives

Several studies have demonstrated an increased risk of adverse health effects, including reduced lung function and lung cancer among asphalt pavers, which has been related to occupational exposure to contaminants during asphalt paving. Consequently, occupational exposure among asphalt pavers must be reduced. The aim of this study was to compare the impact of hot mix asphalt (HMA) and warm mix asphalt (WMA) paving on occupational exposure levels during road paving in field experiments. Asphalt temperatures when paving with WMA are usually lower than when paving with HMA due to differences in the asphalt's composition and method of application.

Methods

On 11 different road sections, one lane was paved with WMA and one with HMA during the same work shift under approximately identical weather conditions. The weather conditions and asphalt surface temperature were monitored during paving. Fifty-seven samples of fumes and vapor, organic and elemental carbon, amines, and respirable, thoracic, and inhalable particulate matter (PM) fractions were collected by stationary sampling. In addition, 30 samples of fumes and vapor were collected by personal sampling.

Results

Compared to paving with HMA, paving with WMA significantly (P < 0.05; paired Student's t-test) reduced the geometric mean (GM) air concentration of asphalt vapor (0.04 versus 0.08 p.p.m.), organic carbon (OC; 0.09 versus 0.18 mg m-3), and respirable PM (0.12 versus 0.22 mg m-3). Additionally, the air concentration of OC correlated strongly with the respirable fraction of PM (Pearson's correlation coefficient 0.83).

Conclusions

Measured airborne concentrations of respirable PM, OC, and asphalt vapor were lower when paving with WMA than with HMA. Because exposure to airborne contaminants generated during asphalt paving is believed to be responsible for the adverse health effects observed among asphalt pavers, paving with WMA rather than HMA may have health benefits.

SUBMITTER: Olsen R

PROVIDER: S-EPMC8091472 | biostudies-literature |

REPOSITORIES: biostudies-literature

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Project description:BackgroundAsphalt workers are exposed to polyaromatic hydrocarbons (PAHs) from hot mix asphalt via both inhalation and dermal absorption. The use of crumb rubber modified (CRM) asphalt may result in higher exposure to PAHs and more adverse effects. Our aim is to assess occupational exposure to PAHs from conventional and CRM asphalt paving by measuring PAH metabolites in urine, and to investigate the effects on mitochondrial DNA copy number (mtDNAcn) and telomere length.MethodsWe recruited 116 workers paving conventional asphalt, 51 workers paving CRM asphalt and 100 controls in Sweden, all males. A repeated-measures analysis included 31 workers paving both types of asphalt. Urine and blood samples were collected pre-working on Monday morning and post-working on Thursday afternoon after 4 days working. PAH metabolites: 1-hydroxypyrene (1-OH-PYR) and 2-hydroxyphenanthrene (2-OH-PH) were measured in urine by LC-MS/MS. Relative mtDNAcn and telomere length were measured by quantitative PCR.ResultsConventional and CRM asphalt workers showed higher 1-OH-PYR and 2-OH-PH than controls (p < 0.001 for all). Relative mtDNAcn were 0.21 units (p < 0.001) higher in conventional asphalt workers and 0.13 units (p = 0.010) higher in CRM asphalt workers compared to controls. Relative telomere length did not differ across occupational groups, but it was positively associated with increment of 2-OH-PH (β = 0.075, p = 0.037) in asphalt workers. The repeated-measures analysis showed no difference in either increment of 1-OH-PYP, or changes in effect biomarkers (mtDNAcn or telomere length) between paving with conventional and CRM asphalt. Increment of 2-OH-PH was smaller after paving with CRM asphalt.ConclusionsRoad asphalt paving in open areas resulted in PAHs exposure, as shown by elevation of PAH metabolites in urine. Asphalt workers may experience oxidative stress, evidenced by alternation in mtDNAcn; however the effects could not be fully explained by exposure to PAHs from the asphalt mixture.