Strategic disinformation outperforms honesty in competition for social influence.
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ABSTRACT:
SUBMITTER: Kurvers RHJM
PROVIDER: S-EPMC8661551 | biostudies-literature |
REPOSITORIES: biostudies-literature
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Project description:The spread of false and misleading information on social media is largely dependent on human action. Understanding the factors that lead social media users to amplify (or indeed intervene in) the spread of this content is an ongoing challenge. Prior research suggests that users are not only more likely to interact with misinformation that supports their ideology or their political beliefs, they may also feel it is more acceptable to spread. However, less is known about the influence of newer, issue-specific beliefs. Two online studies explored the relationship between the degree of belief-consistency of disinformation on users' moral judgements and intentions to spread disinformation further. Four disinformation narratives were presented: disinformation that supported or undermined the UK Government's handling of COVID-19, and disinformation that minimised or maximised the perceived risk of COVID-19. A novel scale for measuring intentions to contribute to the spread of social media content was also used in study 2. Participants reported greater likelihood of spreading false material that was consistent with their beliefs. More lenient moral judgements related to the degree of belief-consistency with disinformation, even when participants were aware the material was false or misleading. These moral judgements partially mediated the relationship between belief-consistency of content and intentions to spread it further on social media. While people are concerned about the spread of disinformation generally, they may evaluate belief-consistent disinformation differently from others in a way that permits them to spread it further. As social media platforms prioritise the ordering of feeds based on personal relevance, there is a risk that users could be being presented with disinformation that they are more tolerant of.
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Project description:Objective: Cortical activity, including cognitive and emotional processes, may influence pupillary function. The exact pathways and the site of cortical pupillary innervation remain elusive, however. We investigated the effects of select cortical strokes, i.e. ischemic infarcts affecting the insular cortex and prefrontal eye field, on pupillary function. Methods: Seventy-four patients with acute ischemic stroke, consecutively admitted to our institution from March to July 2018, were assessed 24 h after endovascular recanalization therapy (i.e., day 2 after the stroke), using automated pupillometry. Stroke location and volume and clinical severity (estimated by the Alberta Stroke Program Early CT Score and National Institute of Health Stroke Scale) were recorded. We excluded patients with posterior circulation stroke, intracranial pathology other than ischemic stroke, midline shift on computed tomography exceeding 5 millimeters or a history of eye disease. Pupillometry data from 25 neurologically normal patients with acute myocardial infarction were acquired for control. Results: Fifty stroke patients after thrombectomy were included for analysis. Twenty-five patients (50%) had insular cortex or prefrontal eye field involvement (group 1, strategic infarcts); 25 patients had infarcts located in other cerebral areas (group 2, other infarcts). The pupillary light reflex, as measured by constriction velocity and maximal/minimal pupillary diameters, was within physiological limits in all patients, including controls. However, while pupillary size and constriction velocities were correlated in all subjects, the correlation of size and dilatation velocity was absent in right-hemispheric infarcts (left hemisphere infarcts, group 1 (r 2 = 0.15, p = 0.04), group 2 (r 2 = 0.41, p = 0.0007); right hemisphere infarcts, group 1 (r 2 = 0.008, p = 0.69); group 2 (r 2 = 0.12, p = 0.08); controls (r 2 = 0.29, p ≤ 0.0001). Conclusions: Cortical infarcts of the prefrontal eye field or insula do not impair the pupillary light reflex in humans. However, subtle changes may occur when the pupils dilate back to baseline, probably due to autonomic dysfunction. Replication is needed to explore the possible influence of hemispheric lateralization. We suggest that endovascular therapy for acute ischemic stroke may serve as a clinical research model for the study of acquired cortical lesions in humans.
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Project description:The medial prefrontal cortex (mPFC) is known as a hub of social hierarchy determination in the brain, Using high throughput single cell transcriptomic analysis and projection-specific genetic manipulation, we discovered that the expression level of Pou3f1 in mPFC-VTA neurons controls social hierarchy.
2023-12-11 | GSE222185 | GEO