Analysis of Claudin 18 deficiency in lung epithelial type 2 cells.
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ABSTRACT: Constitutive knockdown of Cldn18 in mice showed lung enlargement and increased proliferation of alveolar epithelial type II (AT2) cells. Lung AT2 cells were isolated from wild-type and knockout mice and subjected to microarray analysis. Results provide insight into the role of Cldn18 in controlling organ size and stem progenitor cells.
ORGANISM(S): Mus musculus
PROVIDER: GSE106233 | GEO | 2018/06/30
REPOSITORIES: GEO
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