Transcriptomics

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Complement C5a/C5a receptor 1 induces renal injury in diabetic kidney disease via mitochondrial metabolic reprogramming


ABSTRACT: In mice, complement C5a and its receptor C5aR1 elicit systemic and local inflammation and drive tissue injury in diabetic kidney disease via altered metabolic flexibility, which can be attenuated by therapy with a specific orally active inhibitor of C5aR1.

ORGANISM(S): Mus musculus

PROVIDER: GSE118089 | GEO | 2019/07/01

REPOSITORIES: GEO

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