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Expression data from parental and SLX4IP-depleted D2.OR cells

ABSTRACT: Loss of SLX4IP elicits the metastatic outgrowth of nonmetastatic breast cancer cells by activating Wnt signaling to induce telomere maintenance mechanism switching We sought to assess genome-wide transcriptomic profiles governed by loss of SLX4IP expression in latent D2.OR breast cancer cells

ORGANISM(S): Mus musculus

PROVIDER: GSE125702 | GEO | 2019/01/27

REPOSITORIES: GEO

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