Pooled CRISPR screening in pancreatic cancer cells implicates co-repressor complexes as a cause of multiple drug resistance via regulation of epithelial-to-mesenchymal transition [ChIP-seq]
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ABSTRACT: We performed ChIP-seq ananlysis on HDAC1 in MiaPaCa2 using two different antibodies (Santa Cruz Biotechnologies sc-81598 HDAC1(10E2) and Invitrogen HDAC1 Polyclonal Antibody PA1-860), two non-targeting scrableled sgRNA controls and two replicates for each.
ORGANISM(S): Homo sapiens
PROVIDER: GSE158540 | GEO | 2021/05/17
REPOSITORIES: GEO
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