Browse
Submit Data
Databases
API
Help

Dataset Information

0 Views

0 Connections

0 Citations

0 Reanalyses

0 Downloads

Omics score: 0

EBF1 and HIF1a ChIPseq in breast cancer cell lines [ChIP-seq]

ABSTRACT: EBF1 and HIF1a ChIPseq in MDA-MB-157 cells under normoxia and hypoxia

ORGANISM(S): Homo sapiens

PROVIDER: GSE195763 | GEO | 2022/07/08

REPOSITORIES: GEO

ACCESS DATA

Json Xml

Dataset's files

Source:

			Action	DRS
		Other

Items per page:

1 - 1 of 1

Similar Datasets

RNAseq in breast cancer cells with EBF1 knockdown

Project description:Transcriptional analysis of MDA-MB-157 and MDA-MB-436 with EBF1 knockdown

2022-07-08 | GSE195764 | GEO

EBF1 and HIF1a ChIPseq in breast cancer cell lines [ChIP-seq]

Project description:EBF1 and HIF1a ChIPseq in breast cancer cell lines [ChIP-seq]

| PRJNA802116 | ENA

POLR3A ChIPseq

Project description:POLR3A ChIPseq in MDA-231 and MDA-LM2 cells

2022-11-25 | GSE140957 | GEO

ATACSeq and genome-wide binding profile of EBF1 and Pax5 to unravel molecular networks driving leukemia in Ebf1+/-Pax5+/- (dHet) B-ALL mice.

Project description:ATACSeq, EBF1 ChIPSeq and Pax5 ChIPSeq of dHet B-ALL, dHet proB and wt proB cells.

2020-10-19 | GSE158671 | GEO

Genome-wide binding profile of EBF1 in B cells.

Project description:EBF1 ChIPSeq of CD19+ B cells

2021-11-02 | GSE183537 | GEO

ChIPseq of PPARbeta/delta in MDA-MB-231luc cells

Project description:ChIPseq of PPARbeta/delta in MDA-MB-231luc cells

| PRJEB2985 | ENA

HIF1A ChIP-seq from HCT116, RKO, A549, and H460 cells in normoxia and hypoxia

Project description:ChIP-seq analysis was used to identify HIF1A binding sites in normoxia and hypoxia in HCT116, RKO, A549, and H460 cells

2021-01-29 | GSE145157 | GEO

Genes induced by partial down-regulation of ARID1A in breast cancer cell

Project description:Development of gene expression signatures for partial downreualtion of ARID1A protein. ARID1A was down-regulated by si-RNA in a breast cancer cell, MDA-MB-157. Subsequently, RNAs was extracted from control siRNA treated cells or partially ARID1A-down regulated cells. cDNAs were synthesized and cRNAs were labeled using low input quick Amp labeling kit (Agilent). Aftere washing and dry, scanned by Agilent Technologies Micorarrray Scanner and calculated using Agilent Feature 10.7.3.1. control siRNA treated MDA-MB-157 breast cancer cells (MOK) and siRNA-midiated ARID1A-downregulated MDA-MB157 breast cancer cells.

2015-09-03 | E-GEOD-72669 | biostudies-arrayexpress

rBC2LCN lectin detects early-stage HER2-positive breast cancer

Project description:We found that MCF-7 and T-47D or MDA-MB-157 and MDA-MB-231 are rBC2LCN-positive or -negative breast cancer cell lines, respectively. To examine a global gene expression comparison between the rBC2LCN-positive and -negative breast cancer cell lines, DNA microarray analysis was performed.

2020-06-08 | GSE139670 | GEO

A comprehensive Proteomic Investigation of Ebf1 Heterozygosity in pro-B lymphocytes utilizing Data Independent Acquisition

Project description:Early B cell factor 1 (EBF1) is one of the key transcription factors required for orchestrating B-cell lineage development. Although studies have shown that Ebf1 haploinsufficiency is involved in the development of leukemia, no study has been conducted that characterizes the global effect of Ebf1 heterozygosity on the proteome of pro-B lymphocytes. Here, we employ both DIA (Data Independent Acquisition) and shotgun DDA (Data Dependent Acquisition) workflows for profiling proteins that are differently expressed between Ebf1+/+ and Ebf1+/- cells. Both DDA and DIA were able to reveal the downregulation of the EBF1 transcription factor in Ebf1+/- pro-B lymphocytes. Further examination of differentially expressed proteins by DIA revealed that, similar to EBF1, the expression of other B-cell lineage regulators, such as TCF3 and Pax5, is also down-regulated in Ebf1 heterozygous cells. Functional DIA analysis of differentially expressed proteins showed that EBF1 heterozygosity resulted in the deregulation of at least 8 transcription factors involved in lymphopoiesis, and to the deregulation of key proteins playing crucial roles in survival, development and differentiation of pro-B lymphocytes.

2017-12-20 | PXD008396 | Pride

OmicsDI is part of the ELIXIR infrastructure

OmicsDI is an Elixir interoperability service. Learn more ›

Tweets

OmicsDI Databases

PRIDE
PeptideAtlas
MassIVE
JPOST Repository
Physiome Model Repository

EGA
EVA
ENA
LINCS
PAXDB
Cell Collective

MetaboLights
Metabolomics Workbench
MetabolomeExpress
GNPS
BioModels
FAIRDOMHub

ArrayExpress
dbGaP
ExpressionAtlas
GEO
NODE

Information

Databases
Help
API
Contact us
Code on GitHub
Terms of use
Submit Data