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Cdt1 overexpression drives colorectal carcinogenesis through origin overlicensing and DNA damage

ABSTRACT: To examine origin firing upon CDT1 overexpression, EdUseq-HU was performed in HCT116 cells with normal expression levels of CDT1 (OFF) and HCT116 cells with CDT1 overexpression (ON)

ORGANISM(S): Homo sapiens

PROVIDER: GSE212710 | GEO | 2023/09/01

REPOSITORIES: GEO

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Cdt1 overexpression drives colorectal carcinogenesis through origin overlicensing and DNA damage

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