Transcriptional analysis of AHLs signal treatment at 30°C
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ABSTRACT: Yersinia pestis, the etiological agent of plague, is able to sense cell density by quorum sensing. The function of quorum sensing in Y. pestis is not clear. Here, the process of AHL quorum sensing was investigated by comparing transcript profiles when two AHL quorum-sensing signals are added in. The strain Δpgm (pigmentation-negative) mutant was called wild type. The two AHLs signals are N-(3-Oxooctanoyl)-L-homoserine lactone and N-Hexanoyl-DL-homoserine lactone.The control consisted of cells grown and treated under the same conditions without added signals.
ORGANISM(S): Yersinia pestis
PROVIDER: GSE30238 | GEO | 2016/01/01
SECONDARY ACCESSION(S): PRJNA155137
REPOSITORIES: GEO
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