Transcriptomics

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Expression in IFN-gamma treated control and Atg5-/- macrophages


ABSTRACT: Type-I (α/β) and -II (γ) interferons (IFN), through an incompletely understood combination of redundant and unique mechanisms, are essential for host resistance to viral infection. We report a requirement for the Atg5-Atg12/Atg16L1 autophagosome elongation complex in IFNγ-mediated control of murine norovirus in macrophages. We use microarrays to compare transcriptional changes induced in control and Atg5 deficient macrophages by IFNγ treatment.

ORGANISM(S): Mus musculus

PROVIDER: GSE34863 | GEO | 2012/01/05

SECONDARY ACCESSION(S): PRJNA150127

REPOSITORIES: GEO

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