Project description:We have employed whole genome microarray expression profiling to identify genes regulated by Sall1 in the kidney. Kidneys at E13.5 were obtained from inducible Sall1 deletion 24 hrs after tamoxifen treatment (2 set).
Project description:We have employed whole genome microarray expression profiling to identify genes regulated by Sall1 in the kidney. Kidneys at E14.5 were obtained from nephron progenitor-specific Sall1 deletion ( 2 sets) and inducible Sall1 deletion 48 hrs after tamoxifen treatment (1 set).
Project description:We have employed whole genome microarray expression profiling to identify genes regulated by Sall1 in the kidney. Six2GFP-positive nephron progenitors and Six2GFP-negative cells were collected from the embryonic kidneys at E15.5 (2 set) , and their expression profiles were compared.
Project description:To further identify gene expression signatures in the niche cells (CD45 negative) during proliferation of Hematopoietic stem cells (LSK), we employed mice whole genome (60K) microarray expression profiling as a discovery platform to identify the up-regulated and down-regulated genes of the niche.
Project description:To identify differentially up or downregulated genes in MCF7_ADR cell compare to MCF7, we have employed whole genome microarray expression profiling. We used microarray to evaluate the up or down regulated genes in doxorubicin resistant MCF7_ADR cells compare to MCF7
Project description:We have employed whole genome microarray expression profiling as a platform to identify AngII -regulated genes sensitive to CsA. VSMC were treated with AngII with or without CsA ( as an inhibitor of the CN/NFAT pathway).
Project description:In order to study the Myc-regulated gene, we have employed whole genome mRNA/ncRNA microarray expression profiling as a discovery platform to identify genes with the potential to be regulated by Myc. Furthermore, we studied whether some Myc-regulated ncRNAs mediated the function of Myc.
Project description:To further explore gene expression in neonatal mice with Usp10 gene knockout, we have employed whole genome microarray expression profiling as a discovery platform to identify the pathways or biological processes with the potential to explain the cause of postnatal death of Usp10 knockout mice. Kidney tissues from Usp10 knockout neonates and their littermates were used to be the samples to explore.