Changes in gene expression in kidneys from Glis2-deficient mice
Ontology highlight
ABSTRACT: Glis2 is a member of the Gli-similar (Glis) subfamily of Kruppel-like zinc finger transcription factors. To obtain insights into the physiological functions of Glis2, mice deficient in Glis2 expression were generated. These mutant mice develop progressive renal disease associated with fibrosis and inflammation. Gene expression profiles were examined to identify changes in gene expression at early stages of progressive renal disease in Glis2 mutant mice. Keywords: Glis2, kidney, fibrosis, nephropathy, inflammation
ORGANISM(S): Mus musculus
PROVIDER: GSE8454 | GEO | 2009/01/01
SECONDARY ACCESSION(S): PRJNA101535
REPOSITORIES: GEO
ACCESS DATA