Ontology highlight
ABSTRACT:
OTHER RELATED OMICS DATASETS IN: MSV000083572
INSTRUMENT(S): LTQ Orbitrap Velos
ORGANISM(S): Homo Sapiens (human)
TISSUE(S): Platelet
DISEASE(S): Cardiovascular System Disease
SUBMITTER: Pierre Fontana
LAB HEAD: Pierre Fontana
PROVIDER: PXD001861 | Pride | 2016-02-22
REPOSITORIES: Pride
Action | DRS | |||
---|---|---|---|---|
2010_09_07_ALS_18_AnZ_A_R1_01.raw | Raw | |||
2010_09_07_ALS_19_AnZ_A_R1_02.raw | Raw | |||
2010_09_07_ALS_20_AnZ_A_R1_03.raw | Raw | |||
2010_09_07_ALS_21_AnZ_A_R1_04.raw | Raw | |||
2010_09_07_ALS_22_AnZ_A_R1_05.raw | Raw |
Items per page: 5 1 - 5 of 229 |
Zufferey Anne A Ibberson Mark M Reny Jean-Luc JL Nolli Séverine S Schvartz Domitille D Docquier Mylène M Xenarios Ioannis I Sanchez Jean-Charles JC Fontana Pierre P
Human genetics 20160216 4
Platelet reactivity (PR) is variable between individuals and modulates clinical outcome in cardiovascular (CV) patients treated with antiplatelet drugs. Although several data point to a genetic control of platelet reactivity, the genes contributing to the modulation of this phenotype are not clearly identified. Integration of data derived from high-throughput technologies may yield novel insights into the molecular mechanisms that govern platelet reactivity. The aim of this study is to identify ...[more]