Dataset Information
Effect of Cln3-deficiency on protein secretion in Dictyostelium
Ontology highlight
ABSTRACT:
INSTRUMENT(S): Q Exactive
ORGANISM(S): Dictyostelium Discoideum (slime Mold)
SUBMITTER: 
Robert Huber
LAB HEAD: Robert Joseph Huber
PROVIDER: PXD004897 | Pride | 2017-04-04
REPOSITORIES: Pride
Dataset's files
| Action | DRS | |||
|---|---|---|---|---|
| Robert-AX3-1-62715_repeat.msf | Msf | |||
| Robert-AX3-1-62715_repeat.raw | Raw | |||
| Robert-AX3-2_62715.msf | Msf | |||
| Robert-AX3-2_62715.raw | Raw | |||
| Robert-AX3-3-62715_repeat.msf | Msf |
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Publications
Loss of Cln3 impacts protein secretion in the social amoeba Dictyostelium.
Cellular signalling 20170329
Neuronal ceroid lipofuscinosis (NCL), also referred to as Batten disease, is the most common form of childhood neurodegeneration. Mutations in CLN3 cause the most prevalent subtype of the disease, which manifests during early childhood and is currently untreatable. The precise function of the CLN3 protein is still not known, which has inhibited the development of targeted therapies. In the social amoeba Dictyostelium discoideum, loss of the CLN3 homolog, Cln3, reduces adhesion during early devel ...[more]
