Dataset Information
Pathophysiology of arterial cardiovascular events in patients with JAK2V617F myeloproliferative neoplasms
Ontology highlight
ABSTRACT:
INSTRUMENT(S): Q Exactive HF-X
ORGANISM(S): Mus Musculus (mouse)
TISSUE(S): Blood Plasma, Extracellular Vesicle
SUBMITTER: 
Valentin SABATET
LAB HEAD: Damarys Loew
PROVIDER: PXD014451 | Pride | 2020-02-17
REPOSITORIES: Pride
Dataset's files
| Action | DRS | |||
|---|---|---|---|---|
| ExperimentDesign.xls | Xls | |||
| H2162BL.msf | Msf | |||
| H2162BL.raw | Raw | |||
| H2164BL.msf | Msf | |||
| H2164BL.raw | Raw |
Items per page: 1 - 5 of 21 |
Publications
Erythrocyte-derived microvesicles induce arterial spasms in JAK2V617F myeloproliferative neoplasm.
The Journal of clinical investigation 20200501 5
Arterial cardiovascular events are the leading cause of death in patients with JAK2V617F myeloproliferative neoplasms (MPNs). However, their mechanisms are poorly understood. The high prevalence of myocardial infarction without significant coronary stenosis or atherosclerosis in patients with MPNs suggests that vascular function is altered. The consequences of JAK2V617F mutation on vascular reactivity are unknown. We observe here increased responses to vasoconstrictors in arteries from Jak2V617F ...[more]
