DDX24 promotes hepatocellular carcinoma progression via NCL dependent signaling pathway
Ontology highlight
ABSTRACT: Accumulating evidence suggests that DEAD-box proteins are essential in RNA metabolism and play pivotal roles in cancer progression. However, the mechanisms underlying how DDX24 drives hepatocellular carcinoma (HCC) remain largely unknown. In this study, we demonstrated that DDX24 was an oncogene and identified DDX24 promoted HCC development via interacting with NCL.
INSTRUMENT(S): Q Exactive
ORGANISM(S): Homo Sapiens (human)
SUBMITTER:
hairun gan
LAB HEAD: Tianze Liu
PROVIDER: PXD023321 | Pride | 2021-01-29
REPOSITORIES: Pride
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