Ontology highlight
ABSTRACT:
INSTRUMENT(S): Q Exactive
ORGANISM(S): Homo Sapiens (human)
TISSUE(S): Cell Suspension Culture, Lung, Cell Culture
DISEASE(S): Lung Small Cell Carcinoma
SUBMITTER: Nicole Woldmar
LAB HEAD: Melinda Rezeli
PROVIDER: PXD029805 | Pride | 2022-10-14
REPOSITORIES: Pride
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20191025_SCLC10_DIA_R1.raw | Raw | |||
20191025_SCLC10_DIA_R2.raw | Raw | |||
20191025_SCLC11_DIA_R1.raw | Raw | |||
20191025_SCLC11_DIA_R2.raw | Raw | |||
20191025_SCLC12_DIA_R1.raw | Raw |
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Clinical and translational medicine 20220901 9
<h4>Background</h4>Small-cell lung cancer (SCLC) molecular subtypes have been primarily characterized based on the expression pattern of the following key transcription regulators: ASCL1 (SCLC-A), NEUROD1 (SCLC-N), POU2F3 (SCLC-P) and YAP1 (SCLC-Y). Here, we investigated the proteomic landscape of these molecular subsets with the aim to identify novel subtype-specific proteins of diagnostic and therapeutic relevance.<h4>Methods</h4>Pellets and cell media of 26 human SCLC cell lines were subjecte ...[more]