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Aortic infection

ABSTRACT: Mycobacterium chelonae as the probable cause of acute contained rupture of a biological composite graft

PROVIDER: PRJEB39759 | ENA |

REPOSITORIES: ENA

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			Action	DRS
	ERR4672762_1.fastq.gz	Fastqsanger.gz
	ERR4672762_2.fastq.gz	Fastqsanger.gz
	ERR4672763_1.fastq.gz	Fastqsanger.gz
	ERR4672763_2.fastq.gz	Fastqsanger.gz
	ERR4672764_1.fastq.gz	Fastqsanger.gz

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Biofilms of non-tuberculous mycobacteria display distinct expression patterns and form an extracellular matrix

Project description:Comparison of transcriptional differences (RNA-seq) between biofilm and planktonic cultures of Mycobacterium chelonae

2020-08-24 | GSE144514 | GEO

Gene expression study in Positron Emission Tomography (PET) positive abdominal aortic aneurysms

Project description:This study aims at identifying genes involved in this metabolic activation potentially related to rupture. A genome-wide transcriptomic analysis was performed on biopsies collected from patients with a Fluorodeoxyglucose (FDG) uptake both in the positive spot (A+Pos) and in a distant negative site of the same aneurysm (A+Neg). These paired biopsies were further compared to samples collected from (abdominal aortic aneurysms) AAA patients with no FDG uptake (A0) in order to discriminate biological alterations associated with FDG uptake, to detect new systemic biomarkers correlated with a higher risk of rupture and to identify new pathways involved in the progression and rupture of AAA).

2015-02-09 | E-MTAB-3175 | biostudies-arrayexpress

Project description:Mycobacterium chelonae Skin Infection

| PRJNA643662 | ENA

[Mycobacterium] chelonae subsp. chelonae strain:DSM 43804

Project description:[Mycobacterium] chelonae subsp. chelonae strain:DSM 43804 Genome sequencing

| PRJNA508902 | ENA

Mycobacterium

Project description:Genomic variability of Mycobacterium chelonae

| PRJNA323571 | ENA

Mycobacteroides chelonae

Project description:Whole genome sequencing of Mycobacterium chelonae chelonae, an opportunistic pathogen

| PRJDB2512 | ENA

Subcellular pathways shared by afflicted patients and mutant mice identify a new drug treatment for aortic aneurysm in Marfan syndrome

Project description:We analyzed differentially expressed genes in smooth muscle cells derived from the thoracic aorta of Marfan Syndrome (MFS) patients and control subjects to identify cell biological mechanisms contributing to thoracic aoritc aneurysm (TAA) development and rupture. These mechanisms were used to identify a potential drug treatment to mitigate TAA progression. We analyzed differentially expressed genes in whole aorta of P16 MFS mice vs WT mice to identify cell biological mechanisms contributing to thoracic aoritc aneurysm (TAA) development and rupture. These mechanisms were used to identify baclofen as a potential drug treatment to mitigate TAA progression. The effect of baclofen on gene expression in WT and MFS was documented in P60 mice that received treatment since P16.

2019-11-18 | GSE128101 | GEO

Mycobacteroides chelonae

Project description:Whole genome sequencing of Mycobacterium chelonae

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Mycobacteroides chelonae

Project description:Mycobacterium chelonae whole genome sequencing and assembly

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Epicardial transplantation of atrial micrografts

Project description:Ischemic heart disease remains the leading cause of mortality and morbidity worldwide despite improved possibilities in medical care. Alongside interventional therapies, such as coronary artery bypass grafting (CABG), adjuvant tissue-engineered and cell-based treatments can provide regenerative improvement. Unfortunately, most of these advanced approaches require multiple lengthy and costly preparation stages without in turn delivering significant clinical benefit. Here, we investigated the effect of epicardial matrix patch-encased atrial appendage micrografts (AAMs) in a mouse model of myocardial infarction. The matrix-covered AAM patches salvaged the myocardium from infarction-induced functional tissue loss. Fibrosis was attenuated, and site-targeted proteomics revealed AAM patch-activated pathways for angiogenesis and cardiogenesis. The matrix component of the graft further supports functional myocardial recovery by ventricular unloading. The composite epicardial matrix graft encasing AAM micrografts delivers effects desirable for adjuvant cardiac therapy preserving functional cardiac tissue and restricting fibrosis after myocardial infarction.

2019-07-23 | MSV000084120 | MassIVE

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