Browse
Submit Data
Databases
API
Help

Dataset Information

0 Views

0 Connections

0 Citations

0 Reanalyses

0 Downloads

Omics score: 0

Fomes fomentarius isolate:W8498

ABSTRACT: Fomes fomentarius isolate:W8498 Transcriptome or Gene expression

PROVIDER: PRJNA1029195 | ENA |

REPOSITORIES: ENA

ACCESS DATA

Json Xml

Dataset's files

Source:

			Action	DRS
	SRR26448500_1.fastq.gz	Fastqsanger.gz
	SRR26448500_2.fastq.gz	Fastqsanger.gz
	SRR26448501_1.fastq.gz	Fastqsanger.gz
	SRR26448501_2.fastq.gz	Fastqsanger.gz
	SRR26448502_1.fastq.gz	Fastqsanger.gz

Items per page:

1 - 5 of 18

Similar Datasets

Fomes fomentarius

Project description:Fomes fomentarius overview

| PRJNA445158 | ENA

Fomes fomentarius strain:CIRM-BRFM 1821 | isolate:CIRM-BRFM 1821

Project description:Fomes fomentarius CIRM-BRFM 1821 transcriptome - 1821_RNA

| PRJNA442214 | ENA

Fomes fomentarius strain:CIRM-BRFM 1821

Project description:Fomes fomentarius CIRM-BRFM 1821 Standard Draft genome sequencing

| PRJNA444027 | ENA

Heritiera fomes isolate:HF-1

Project description:Heritiera fomes isolate:HF-1 Genome sequencing

| PRJNA783468 | ENA

Heritiera fomes

Project description:Heritiera fomes overview

| PRJNA804993 | ENA

Project description:Transcriptomics of Fomes fomentarius on various substrates

| PRJNA957595 | ENA

METAGENOMIC ANALYSIS OF HERITIERA FOMES

Project description:METAGENOMIC ANALYSIS OF HERITIERA FOMES Raw sequence reads

| PRJNA689209 | ENA

Project description:Metagenomics and metatranscriptomics of fresh and rotting Fomes fomentarius fruiting bodies

| PRJNA1037776 | ENA

Loss of sterol demethylase activity reduces both azole and echinocandin antifungal susceptibility in a clinical isolate of C. parapsilosis

Project description:The present study describes a novel mechanism of antifungal resistance affecting the susceptibility of both the azole and echinocandin antifungals in an azole-resistant isolate from a matched pair of C. parapsilosis isolates obtained from a patient with prosthetic valve endocarditis. Transcriptome analysis indicated differential expression of several genes in the resistant isolate including upregulation of ERG1, ERG2, ERG5, ERG6, ERG11, ERG24, ERG25, ERG27, DAP1 and UPC2, of the ergosterol biosynthesis pathway. Whole genome sequencing revealed a mutation in the ERG3 gene leading to a G111R amino acid substitution in the resistant isolate. Subsequent introduction of this allele in the native ERG3 locus in the susceptible isolate resulted in a fluconazole MIC of >64 mg/ml and a caspofungin MIC of 8 mg/ml. Corresponding allelic replacement of the wildtype allele for the mutant allele in the resistant isolate resulted in a drop in MIC to 1 mg/ml for both fluconazole and caspofungin. Sterol profiles indicated a loss of sterol demethylase activity as a result of this mutation. This work demonstrate that this G111R mutation is wholly responsible for the resistant phenotype in the C. parapsilosis resistant isolate and is the first report of this multidrug resistance mechanism.

2017-05-25 | GSE98986 | GEO

Next Generation Sequencing of Wildtype and Thioredoxin-A knockout of a clinical isolate of Acinetobacter baumannii

Project description:RNA sequencing was used to analyze the transcriptome of a thioredoxin-A knockout of a clinical isolate of Acinetobacter baumannii

2019-01-14 | GSE125017 | GEO

OmicsDI is part of the ELIXIR infrastructure

OmicsDI is an Elixir interoperability service. Learn more ›

Tweets

OmicsDI Databases

PRIDE
PeptideAtlas
MassIVE
JPOST Repository
Physiome Model Repository

EGA
EVA
ENA
LINCS
PAXDB
Cell Collective

MetaboLights
Metabolomics Workbench
MetabolomeExpress
GNPS
BioModels
FAIRDOMHub

ArrayExpress
dbGaP
ExpressionAtlas
GEO
NODE

Information

Databases
Help
API
Contact us
Code on GitHub
Terms of use
Submit Data