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ATAC-seq of mouse cortex undergoing early degradation after traumatic brain injury (TBI)

ABSTRACT: ATAC-seq of mouse cortex undergoing early degradation after traumatic brain injury (TBI)

PROVIDER: PRJNA1029568 | ENA |

REPOSITORIES: ENA

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			Action	DRS
	SRR26421786_1.fastq.gz	Fastqsanger.gz
	SRR26421786_2.fastq.gz	Fastqsanger.gz
	SRR26421787_1.fastq.gz	Fastqsanger.gz
	SRR26421787_2.fastq.gz	Fastqsanger.gz
	SRR26421788_1.fastq.gz	Fastqsanger.gz

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ATAC-seq of mouse cortex undergoing early degradation after traumatic brain injury (TBI)

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Proteomics characterization of the neuroprotective effects of low-dose methamphetamine on rats with traumatic brain injury (TBI)

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Chronic Gene Expression after Traumatic Brain Injury

Project description:This project continues our acute TBI studies and initiates chronic studies -- characterizing the temporal genomic profile of the injured brain up to 12 months post-TBI. Thus, the primary objective of this proposal is to determine the long-term effects of traumatic brain injury on gene expression in the hippocampus and cortex, two brain regions known to be particularly vulnerable to TBI.

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