Browse
Submit Data
Databases
API
Help

Dataset Information

0 Views

0 Connections

0 Citations

0 Reanalyses

0 Downloads

Omics score: 0

ChIP-seq of NRF1 in skeletal muscle

ABSTRACT: ChIP-seq of NRF1 in skeletal muscle

PROVIDER: PRJNA1060641 | ENA |

REPOSITORIES: ENA

ACCESS DATA

Json Xml

Dataset's files

Source:

			Action	DRS
	SRR27407600_1.fastq.gz	Fastqsanger.gz
	SRR27407600_2.fastq.gz	Fastqsanger.gz
	SRR27407599_1.fastq.gz	Fastqsanger.gz
	SRR27407599_2.fastq.gz	Fastqsanger.gz

Items per page:

1 - 4 of 4

Similar Datasets

ChIP-seq of NRF1 in skeletal muscle

Project description:Charaterize NRF1 cistrome in mouse muscle

2024-03-22 | GSE252431 | GEO

Epigenetic changes during skeletal muscle atroph [ChIP-seq]

Project description:Epigenetic changes during skeletal muscle atroph [ChIP-seq]

| PRJNA899567 | ENA

Epigenetic changes during skeletal muscle atroph [ChIP-seq]

Project description:The epigenomic regulation is a part of Gene Regulatory Network (GRN). During we study the reprogramming of GRN adaptive to atrophic stimulation in skeletal muscle, we performed Histone 3 lysine 27 (H3K27) acetylation (H3K27ac) ChIP-seq assay using mouse skeletal muscle with or without denervation. This dataset combining with our snATAC datasets enable us to infer the candidate enhancer that could regulate muscle protein metabolism and energy metabolism during atrophy.

2023-09-06 | GSE217574 | GEO

Skeletal muscle myonuclei-specific ChIP-Seq of H3K4me3 and H3K27ac

Project description:Skeletal muscle myonuclei-specific ChIP-Seq of H3K4me3 and H3K27ac

| PRJNA757393 | ENA

Control ChIP-seq from skeletal muscle myoblast (ENCSR000ANN)

Project description:Control ChIP-seq on human skeletal muscle myoblast For data usage terms and conditions, please refer to http://www.genome.gov/27528022 and http://www.genome.gov/Pages/Research/ENCODE/ENCODE_Data_Use_Policy_for_External_Users_03-07-14.pdf

2019-08-30 | GSE136426 | GEO

Global MEF2 target gene analysis in cardiac and skeletal muscle reveals novel regulation of DUSP6 by p38MAPKMEF2 signaling [ChIP-seq]

Project description:Identfification of MEF2A target genes using ChIP-exo in skeletla muscle and primary cardiomyocytes. Identfification of MEF2A target genes using ChIP-exo and RNA-seq in skeletal muscle and primary cardiomyocytes. MEF2 plays a profound role in the regulation of transcription in cardiac and skeletal muscle lineages. To define the overlapping and unique MEF2A genomic targets, we utilized ChIP-exo analysis of cardiomyocytes and skeletal myoblasts. Of the 2783 and 1648 MEF2A binding peaks in skeletal myoblasts and cardiomyocytes, respectively, 294 common binding sites were identified. Genomic targets were compared to differentially expressed genes in RNA-seq analysis of MEF2A depleted myogenic cells. MEF2A target genes were identified in 48 hr DM C2C12 myoblasts cells and primary cardiomyocytes using ChIP-exo. Binding profiles on MEF2A in each cell type were compared. Cross sectional-analysis between ChIP-exo identified targets and RNA-seq analysis of MEF2A deplted myoblasts was also done.

2014-09-09 | E-GEOD-61204 | biostudies-arrayexpress

NURR1 activation in skeletal muscle controls systemic energy homeostasis (ChIP-seq)

Project description:NURR1 activation in skeletal muscle controls systemic energy homeostasis (ChIP-seq)

| PRJNA541537 | ENA

ChIP-seq of GATA4 in mouse skeletal muscle myoblasts

Project description:GATA4 occupancy on the mouse genome of satellite cell-derived primary myoblasts. Proliferating myoblasts cultured in growth medium were immunoprecipitated with anti-GATA4 antibody or control IgG. Precipitated genomic DNAs were subjected to next generation sequencing. Paired-end 150 bp sequence reads of GATA4-ChIP and IgG-ChIP using mouse skeletal muscle myoblasts.

2015-04-10 | E-GEOD-67703 | biostudies-arrayexpress

10X skeletal muscle regeneration scRNA-seq

Project description:10X skeletal muscle regeneration scRNA-seq

| PRJNA577458 | ENA

ATAC-seq in mouse skeletal muscle

Project description:ATAC-seq in mouse skeletal muscle

| PRJNA637258 | ENA