Browse
Submit Data
Databases
API
Help

Dataset Information

0 Views

0 Connections

0 Citations

0 Reanalyses

0 Downloads

Omics score: 0

Ureibacillus massiliensis 4400831 = CIP 108448 = CCUG 49529

ABSTRACT: Ureibacillus massiliensis 4400831 = CIP 108448 = CCUG 49529 Genome sequencing and assembly

PROVIDER: PRJNA214581 | ENA |

REPOSITORIES: ENA

ACCESS DATA

Json Xml

Dataset's files

Source:

			Action	DRS
		Other

Items per page:

1 - 1 of 1

Similar Datasets

Ureibacillus massiliensis 4400831 = CIP 108448 = CCUG 49529

Project description:Ureibacillus massiliensis 4400831 = CIP 108448 = CCUG 49529 Genome sequencing and assembly

| PRJNA239540 | ENA

Staphylococcus massiliensis CCUG 55927

Project description:Staphylococcus massiliensis CCUG 55927 Genome sequencing and assembly

| PRJNA165175 | ENA

Haematobacter massiliensis strain:CCUG 47968

Project description:Haematobacter massiliensis strain:CCUG 47968 Genome sequencing and assembly

| PRJNA241047 | ENA

Ureibacillus massiliensis

Project description:Lysinibacillus massiliensis overview

| PRJNA169380 | ENA

p/CIP gene regulation in mouse embryonic stem cells

Project description:p/CIP binds to many nuclear receptors and plays a major role in hormone dependent transcription of genes. Recently, p/CIP was shown to affect mouse stem cell pluripotency. Microarray gene expression analysis was conducted to assess the role of p/CIP in mouse embryonic stem cells.

2013-08-21 | GSE50017 | GEO

Ureibacillus thermosphaericus

Project description:Ureibacillus thermosphaericus overview

| PRJNA89573 | ENA

Transcription analysis of E. coli O103:H25 in co-culture with Bacteroides thetaiotaomicron CCUG 10774

Project description:Investigation of whole genome gene expression level changes in E. coli O103:H25 in co-culture with Bacteroides thetaiotaomicron CCUG 10774 compared to when E. coli is cultured individually.

2014-12-18 | GSE44790 | GEO

CIP, a cardioprotective factor, inhibits the transition from cardiac hypertrophy to heart failure

Project description:Heart failure is characterized by the inability of the heart to pump effectively and generate proper blood circulation to meet the body's needs; it is a devastating condition that affects more than 100 million people globally. In spite of this, little is known about the mechanisms regulating the transition from cardiac hypertrophy to heart failure. Previously, we identified a cardiomyocyte-enriched gene, CIP, which regulates cardiac homeostasis under pathological stimulation. Here, we show that the cardiac transcriptional factor GATA4 binds the promotor of CIP gene and regulates its expression. We further determined that both CIP mRNA and protein decrease in diseased human hearts. In a mouse model, induced cardiac-specific overexpression of CIP after the establishment of cardiac hypertrophy protects the heart by inhibiting disease progression toward heart failure. Transcriptome analyses revealed that the IGF, mTORC2 and TGFβ signaling pathways mediate the inhibitory function of CIP on pathologic cardiac remodeling. Our study demonstrates GATA4 as an upstream regulator of CIP gene expression in cardiomyocytes, as well as the clinical significance of CIP expression in human heart disease. More importantly, our investigation suggests CIP is a key regulator of the transition from cardiac hypertrophy to heart failure. The ability of CIP to intervene in the onset of heart failure suggests a novel therapeutic avenue of investigation forthe prevention of heart disease progression.

2022-03-05 | GSE194149 | GEO

Ureibacillus manganicus DSM 26584

Project description:Ureibacillus manganicus DSM 26584 Genome sequencing and assembly

| PRJNA239539 | ENA

Ureibacillus manganicus DSM 26584

Project description:Ureibacillus manganicus DSM 26584 Genome sequencing and assembly

| PRJNA214585 | ENA