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GR cistromes reprogramming by High Fat Diet [ChIP-seq H3K27ac]

ABSTRACT: GR cistromes reprogramming by High Fat Diet [ChIP-seq H3K27ac]

PROVIDER: PRJNA530560 | ENA |

REPOSITORIES: ENA

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Dataset's files

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			Action	DRS
	SRR8837520_1.fastq.gz	Fastqsanger.gz
	SRR8837520_2.fastq.gz	Fastqsanger.gz
	SRR8837521_1.fastq.gz	Fastqsanger.gz
	SRR8837521_2.fastq.gz	Fastqsanger.gz
	SRR8837522_1.fastq.gz	Fastqsanger.gz

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GR cistromes reprogramming by High Fat Diet [ChIP-seq: GR DEX]

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GR cistromes reprogramming by High Fat Diet [ChIP-seq H3K27ac]

Project description:We mapped the genome-wide profiles of Histone H3K27 acetylation (two time points, ZT0 and ZT12) by using ChIP-Seq in livers from control mice and mice fed High Fat Diet (HFD) for 12 weeks

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Identify acetylation marks in adult mouse L4 vertebrae gene promoter regions from maternal high fat diet dams using H3K27ac ChIP-Seq

Project description:Genes with increased H3K27ac marks in their promoters were found in adult mice L4 vertebrae from mice using H3K27ac ChIP-Seq. Recent data suggests that in animals and humans that a maternal diet high in fat leads to poor bone growth and development in offspring. Genes with increased H3K27ac epigenetic marks normally have higher levels of expression. Understanding which genes are being upregulated due to maternal or offspring high fat diet would allow for better treatments to combat bone loss to be developed.

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Genome-wide profiling of H3K27ac, H3K4me1, H3K27me3 and H3K9me3 (ChIP-seq) in U2OS-GR and U2OS-AR cells

Project description:We performed ChIP-seq targeting the H3K27ac, H3K4me1, H3K27me3 and H3K9me3 in the U2OS-GR and U2OS-AR cell lines. The cell lines are derived from U2OS ATTC:HTB-96 and stably transfected with an expression construct for either rat GR or human AR, respectively. The U2OS-GR cells were treated with dexamethasone (1 µM) or vehicle (ethanol) for 90 minutes. The U2OS-AR cells were treated with R1881 (5 nM) or vehicle (DMSO) for 4 hours.

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2022-05-31 | E-MTAB-11712 | biostudies-arrayexpress

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