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Ctenopharyngodon idella

ABSTRACT: GCRV infection CIK cells

PROVIDER: PRJNA579880 | ENA |

REPOSITORIES: ENA

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			Action	DRS
	SRR10389200_1.fastq.gz	Fastqsanger.gz
	SRR10389200_2.fastq.gz	Fastqsanger.gz
	SRR10389201_1.fastq.gz	Fastqsanger.gz
	SRR10389201_2.fastq.gz	Fastqsanger.gz

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Ctenopharyngodon idella

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Project description:The destiny of the resistance/susceptibility against GCRV has been congenitally destined by epigenetic mechanism in CIK cells [miRNA-seq]

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Project description:The destiny of the resistance/susceptibility against GCRV has been congenitally destined by epigenetic mechanism in CIK cells (MeDIP-seq)

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Project description:The destiny of the resistance/susceptibility against GCRV has been congenitally destined by epigenetic mechanism in CIK cells [RNA-seq]

| PRJNA344676 | ENA

MeDIP-seq of Ctenopharyngodon idellus kidney cells after grass carp reovirus infection

Project description:MeDIP-seq of Ctenopharyngodon idellus kidney cells after GCRV infected 8 hours.

2024-05-11 | E-MTAB-13003 | biostudies-arrayexpress

RNA-seq of Ctenopharyngodon idellus kidney cells after grass carp reovirus infection

Project description:RNA-seq of Ctenopharyngodon idellus kidney cells after GCRV infected 4hours and 8 hours.

2024-05-11 | E-MTAB-13002 | biostudies-arrayexpress

The destiny of the resistance/susceptibility against GCRV has been congenitally destined by epigenetic mechanism in CIK cells [miRNA-seq]

Project description:In this study, the resistant and susceptible monoclonal CIK cell lines were first established which would be the ponderable research models for the nosogenesis mechanism of the hemorrhagic disease. C1 (CIK cells), R2 (resistant cells) and S3 (susceptible cells) samples were carried out RNA-Seq, MeDIP-Seq and small RNA-Seq by the next-generation sequencing strategy, bioinformatics analysis as well as experimental verification. It was discovered that the aboriginality of CIK cells were gravitated to the susceptible trait. And the discrepancies between resistance and susceptibility against GCRV could primarily attribute to antioxidant activity, cell killing activity and cell proliferation regulation. Here we comprehensively present the profiling and characteristics of DNA methylation and microRNA in the resistant and susceptible CIK cells and proposed that high mCHH methylation distribution might be a characteristic modulator in C. idella. What’s more, a series of genes modulated by DNA methylation or microRNA were designated as potential biomarkers for the resistance breeding. This study laid the foundation and opened novel avenues for nosogenesis research on hemorragic disease of C. idella.

2016-09-28 | GSE87413 | GEO

Ctenopharyngodon idella, CIK cells, pull-down MS

Project description:IL6-STAT3 is a canonical pro-inflammatory pathway that is highly related to temperature fluctuation and viral infection. Our understanding concerning how the crosstalk between IL6-STAT3 signaling axis and environmental temperature determines viral infection remains rudimentary. Employing an aquatic reovirus infection in ectotherms as a model system, we delineated a pivotal role of IL6-STAT3-HSP90 signaling axis in temperature dependent pathogenesis through interacting with viral capsid proteins to promote viral entry.

2023-05-09 | PXD036006 | Pride

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