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High throughput sequencing in ARID1A knockout MDA-MB-231 cells [H3K27ac histone modification ChIP-seq]

ABSTRACT: High throughput sequencing in ARID1A knockout MDA-MB-231 cells [H3K27ac histone modification ChIP-seq]

PROVIDER: PRJNA994421 | ENA |

REPOSITORIES: ENA

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			Action	DRS
	SRR25255805_1.fastq.gz	Fastqsanger.gz
	SRR25255805_2.fastq.gz	Fastqsanger.gz
	SRR25255806_1.fastq.gz	Fastqsanger.gz
	SRR25255806_2.fastq.gz	Fastqsanger.gz
	SRR25255807_1.fastq.gz	Fastqsanger.gz

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High throughput sequencing in ARID1A knockout MDA-MB-231 cells [H3K27ac histone modification ChIP-seq]

Project description:ARID1A has been suggested as a key regulator for anti-tumor immunity. In the present study, we evaluated the role of ARID1A deficiency in inducing adaptive immune resistance in triple negative breast cancer. To explore global transcriptional activity, we performed chromatin immunoprecipitation followed by high throughput sequencing (ChIP–seq) to investigate H3K27ac histone modification of ARID1A knockout and control MDA-MB-231 cells.

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Project description:ARID1A has been suggested as a key regulator for anti-tumor immunity. In the present study, we evaluated the role of ARID1A deficiency in inducing adaptive immune resistance in triple negative breast cancer. To explore global transcriptional activity, we performed chromatin immunoprecipitation followed by high throughput sequencing (ChIP–seq) to investigate H3K4me1 histone modification of ARID1A knockout and control MDA-MB-231 cells.

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The miRNA profiling in MDA-MB-231, MDA-MB-231/E1A and knockdown of Dicer in MDA-MB-231/E1A cells

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High-throughput sequencing in ARID1A knockout MDA-MB-231 cells

Project description:High-throughput sequencing in ARID1A knockout MDA-MB-231 cells

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