Ontology highlight
ABSTRACT:
SUBMITTER: Earp M
PROVIDER: S-EPMC4884925 | biostudies-literature | 2016 Feb
REPOSITORIES: biostudies-literature
Earp Madalene M Winham Stacey J SJ Larson Nicholas N Permuth Jennifer B JB Sicotte Hugues H Chien Jeremy J Anton-Culver Hoda H Bandera Elisa V EV Berchuck Andrew A Cook Linda S LS Cramer Daniel D Doherty Jennifer A JA Goodman Marc T MT Levine Douglas A DA Monteiro Alvaro N A AN Ness Roberta B RB Pearce Celeste L CL Rossing Mary Anne MA Tworoger Shelley S SS Wentzensen Nicolas N Bisogna Maria M Brinton Louise L Brooks-Wilson Angela A Carney Michael E ME Cunningham Julie M JM Edwards Robert P RP Fogarty Zachary C ZC Iversen Edwin S ES Kraft Peter P Larson Melissa C MC Le Nhu D ND Lin Hui-Yi HY Lissowska Jolanta J Modugno Francesmary F Moysich Kirsten B KB Olson Sara H SH Pike Malcolm C MC Poole Elizabeth M EM Rider David N DN Terry Kathryn L KL Thompson Pamela J PJ van den Berg David D Vierkant Robert A RA Vitonis Allison F AF Wilkens Lynne R LR Wu Anna H AH Yang Hannah P HP Ziogas Argyrios A Phelan Catherine M CM Schildkraut Joellen M JM Chen Yian Ann YA Sellers Thomas A TA Fridley Brooke L BL Goode Ellen L EL
Oncotarget 20160201 7
<h4>Background</h4>Genome-wide association studies have identified several common susceptibility alleles for epithelial ovarian cancer (EOC). To further understand EOC susceptibility, we examined previously ungenotyped candidate variants, including uncommon variants and those residing within known susceptibility loci.<h4>Results</h4>At nine of eleven previously published EOC susceptibility regions (2q31, 3q25, 5p15, 8q21, 8q24, 10p12, 17q12, 17q21.31, and 19p13), novel variants were identified t ...[more]