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Protection of the patient data against intentional attacks using a hybrid robust watermarking code.

ABSTRACT: The security of patient information is important during the transfer of medical data. A hybrid spatial domain watermarking algorithm that includes encryption, integrity protection, and steganography is proposed to strengthen the information originality based on the authentication. The proposed algorithm checks whether the patient's information has been deliberately changed or not. The created code is distributed at every pixel of the medical image and not only in the regions of non-interest pixels, while the image details are still preserved. To enhance the security of the watermarking code, SHA-1 is used to get the initial key for the Symmetric Encryption Algorithm. The target of this approach is to preserve the content of the image and the watermark simultaneously, this is achieved by synthesizing an encrypted watermark from one of the components of the original image and not by embedding a watermark in the image. To evaluate the proposed code the Least Significant Bit (LSB), Bit2SB, and Bit3SB were used. The evaluation of the proposed code showed that the LSB is of better quality but overall the Bit2SB is better in its ability against the active attacks up to a size of 2*2 pixels, and it preserves the high image quality.

SUBMITTER: Nagm A

PROVIDER: S-EPMC8022583 | biostudies-literature |

REPOSITORIES: biostudies-literature

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Project description:Bacterial competition may rely on secretion systems such as the type 6 secretion system (T6SS), which punctures and releases toxic molecules into neighboring cells. To subsist, bacterial targets must counteract the threats posed by T6SS-positive competitors. In this study, we used a comprehensive genome-wide high-throughput screening approach to investigate the dynamics of interbacterial competition. Our primary goal was to identify deletion mutants within the well-characterized E. coli K-12 single-gene deletion library, the Keio collection, that demonstrated resistance to T6SS-mediated killing by the enteropathogenic bacterium Cronobacter malonaticus. We identified 49 potential mutants conferring resistance to T6SS and focused our interest on a deletion mutant (∆fimE) exhibiting enhanced expression of type 1 fimbriae. We demonstrated that the presence of type 1 fimbriae leads to the formation of microcolonies and thus protects against T6SS-mediated assaults. Collectively, our study demonstrated that adhesive structures such as type 1 fimbriae confer collective protective behavior against T6SS attacks.IMPORTANCEType 6 secretion systems (T6SS) are molecular weapons employed by gram-negative bacteria to eliminate neighboring microbes. T6SS plays a pivotal role as a virulence factor, enabling pathogenic gram-negative bacteria to compete with the established communities to colonize hosts and induce infections. Gaining a deeper understanding of bacterial interactions will allow the development of strategies to control the action of systems such as the T6SS that can manipulate bacterial communities. In this context, we demonstrate that bacteria targeted by T6SS attacks from the enteric pathogen Cronobacter malonaticus, which poses a significant threat to infants, can develop a collective protective mechanism centered on the production of type I fimbriae. These adhesive structures promote the aggregation of bacterial preys and the formation of microcolonies, which protect the cells from T6SS attacks.

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Protection of the patient data against intentional attacks using a hybrid robust watermarking code.

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