Basal-to-inflammatory transition enhances basal cell carcinoma (BCC) therapy resistance via crosstalk with a pro-inflammatory stromal niche [RNA-seq]
Ontology highlight
ABSTRACT: The goal of these RNA-seq experiments was to determine transcriptomic gene expression changes caused by Il1 or Osm (or both) treatments of mouse BCC cells at 24h and 48h timepoints versus PBS control
ORGANISM(S): Mus musculus
PROVIDER: GSE254600 | GEO | 2024/07/26
REPOSITORIES: GEO
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