Basal-to-inflammatory transition enhances basal cell carcinoma (BCC) therapy resistance via crosstalk with a pro-inflammatory stromal niche [RNA-seq II]
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ABSTRACT: The goal of these RNA-seq experiments was to determine transcriptomic gene expression changes caused by 7 days of Il1a and Osm combination treatment of mouse primary BCC tumor-derived organoids compared to PBS control
ORGANISM(S): Mus musculus
PROVIDER: GSE273014 | GEO | 2024/07/26
REPOSITORIES: GEO
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