Browse
Submit Data
Databases
API
Help

Dataset Information

0 Views

0 Connections

0 Citations

0 Reanalyses

0 Downloads

Omics score: 0

Gene expression profiles of Hepa1-6 tumors in C57Bl/6 mice

ABSTRACT: Chronological change of gene expression profile of Hepa1-6 tumors in C57Bl/6

ORGANISM(S): Mus musculus

PROVIDER: GSE38981 | GEO | 2012/07/03

SECONDARY ACCESSION(S): PRJNA169682

REPOSITORIES: GEO

ACCESS DATA

Json Xml

Dataset's files

Source:

			Action	DRS
		Other

Items per page:

1 - 1 of 1

Similar Datasets

Gene expression profiles of Hepa1-6 tumors in C57Bl/6 mice

Project description:Chronological change of gene expression profile of Hepa1-6 tumors in C57Bl/6 Two-condition experiment

2012-07-02 | E-GEOD-38981 | biostudies-arrayexpress

Gene expression profiles of whole blood cells of C57Bl/6 mice with Hepa1-6 tumors

Project description:Chronological change of gene expression profile of whole blood cells of C57Bl/6 with Hepa1-6 tumors

2012-07-03 | GSE38980 | GEO

Gene expression profiles of whole blood cells of C57Bl/6 mice with Hepa1-6 tumors

Project description:Chronological change of gene expression profile of whole blood cells of C57Bl/6 with Hepa1-6 tumors Two-condition experiment

2012-07-02 | E-GEOD-38980 | biostudies-arrayexpress

Gene expression profiles of whole blood cells and the fraction enriched with tumor-infiltrating inflammatory cells of Balb/c athymic mice with Hepa1-6 tumors

Project description:Chronological change of gene expression profile of whole blood cells and features of the fraction enriched with tumor-infiltrating inflammatory cells of Balb/c athymic mice with Hepa1-6 tumors Two-condition experiment

2012-07-02 | E-GEOD-38976 | biostudies-arrayexpress

Gene expression profiles of whole blood cells and the fraction enriched with tumor-infiltrating inflammatory cells of Balb/c athymic mice with Hepa1-6 tumors

2012-07-03 | GSE38976 | GEO

Stable overexpression of IRF8 in hepatocellular carcinoma cells hepa1-6 xenograft derived tumors in C57bl/6 mice

Project description:Stable overexpression of IRF8 in hepatocellular carcinoma cells hepa1-6 xenograft derived tumors in C57bl/6 mice

| PRJNA700621 | ENA

Expression data from syngeneic Hepa1-6 model in C57Bl/6J mice

Project description:Expression data from syngeneic Hepa1-6 model in C57Bl/6J mice

| PRJNA641716 | ENA

Gene expression analysis of blood cells and tumor

Project description:This SuperSeries is composed of the following subset Series: GSE38976: Gene expression profiles of whole blood cells and the fraction enriched with tumor-infiltrating inflammatory cells of Balb/c athymic mice with Hepa1-6 tumors GSE38979: Gene expression profiles of blood and the enriched fraction of tumor-infiltrating inflammatory cells of C57Bl/6 mice with Hepa1-6 tumors GSE38980: Gene expression profiles of whole blood cells of C57Bl/6 mice with Hepa1-6 tumors GSE38981: Gene expression profiles of Hepa1-6 tumors in C57Bl/6 mice Refer to individual Series

2012-07-02 | E-GEOD-39075 | biostudies-arrayexpress

Expression data from syngeneic Hepa1-6 model in C57Bl/6J mice

Project description:Lenvatinib is an effective drug in advanced hepatocellular carcinoma (HCC). Its combination with the anti-PD1 immune checkpoint inhibitor pembrolizumab has achieved encouraging results in phase Ib and is currently being tested in phase III trials. Here, we aimed at exploring the molecular and immunomodulatory effects of lenvatinib alone or in combination with anti-PD1. We generated a syngeneic model of HCC in C57BL/6J mice and randomized the animals to receive placebo, lenvatinib, anti-PD1 or combination treatment. Transcriptomic analysis were performed to assess the expression profile of tumors from mice receiving each treatment strategy.

2023-04-07 | GSE153203 | GEO

Transcriptional profiling of Hepa1-6-bearing mouse model

Project description:Hepa1-6-bearing C57BL/6 mouse model was established to evaluate the therapeutic efficacy of Ganoderma lucidum extract (GLE) in HCC. The GLE treated and untreated mice were used for transcriptioanl profiles detection, and 126 differentially expressed lncRNAs and 558 DE mRNAs were identified, respectively. The bioinformatics analysis shows that the GLE could suppress the growth and proliferation of HCC by PI3K/Akt/mTOR and MAPK signaling pathway, and also could induce apoptosis of tumor cell by mitochondrial and death receptor pathway.

2018-08-02 | GSE117503 | GEO

OmicsDI is part of the ELIXIR infrastructure

OmicsDI is an Elixir interoperability service. Learn more ›

Tweets

OmicsDI Databases

PRIDE
PeptideAtlas
MassIVE
JPOST Repository
Physiome Model Repository

EGA
EVA
ENA
LINCS
PAXDB
Cell Collective

MetaboLights
Metabolomics Workbench
MetabolomeExpress
GNPS
BioModels
FAIRDOMHub

ArrayExpress
dbGaP
ExpressionAtlas
GEO
NODE

Information

Databases
Help
API
Contact us
Code on GitHub
Terms of use
Submit Data