Effects of VEGF-B on doxorubicin-induced cardiotoxicity
Ontology highlight
ABSTRACT: Doxorubicin is a widely used and effective anthracycline chemotherapy drug, which use is limited due to its cardiotoxic side effects. We show that gene therapy with AAV-VEGF-B can inhibit the doxorubicin-induced cardiac atrophy and whole body cachexia.
ORGANISM(S): Mus musculus
PROVIDER: GSE81448 | GEO | 2016/09/30
SECONDARY ACCESSION(S): PRJNA321605
REPOSITORIES: GEO
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