JunB is essential for IL-23-dependent pathogenicity of Th17 cells.
Ontology highlight
ABSTRACT: To understand molecular mechanisms by which JunB regulates Th17 differentiation, we performed microarray analyses of JunB-deficient and control Th17 cells.
ORGANISM(S): Mus musculus
PROVIDER: GSE86499 | GEO | 2017/04/11
SECONDARY ACCESSION(S): PRJNA342088
REPOSITORIES: GEO
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