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GNPS GCMS Inhibition of mitochondrial respiration LCMS data

ABSTRACT: Description Mitochondrial respiration in mammalian cells not only generates ATP to meet their own energy needs but also couples with biosynthetic pathways to produce metabolites that can be exported to support neighboring cells. However, how defects in mitochondrial respiration influence these biosynthetic and exporting pathways remains poorly understood. In this study we used targeted-metabolomics to investigate how inhibition of mitochondrial respiration influences the intracellular and extracellular metabolome.

INSTRUMENT(S): Agilent 7890B/5977B GC MS system with a DB-5MS column

ORGANISM(S): Homo Sapiens (ncbitaxon:9606)

SUBMITTER: Jianhai Du

PROVIDER: MSV000086159 | GNPS |

REPOSITORIES: GNPS

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