Proteomics

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CPTAC proteomic analysis of TCGA colon and rectal carcinomas using standard and customized databases, part 2

ABSTRACT: The goal of the CPTAC, TCGA Cancer Proteome Study of Colorectal Tissue is to analyze the proteomes of TCGA tumor samples that have been comprehensively characterized by molecular methods. Ninety-five TCGA tumor samples were used in this study.

INSTRUMENT(S): LTQ Orbitrap Velos

ORGANISM(S): Homo Sapiens (human)

TISSUE(S): Rectal Cancer Cell, Colonic Cancer Cell

DISEASE(S): Colon Adenocarcinoma,Rectum Adenocarcinoma

SUBMITTER: Robbert Slebos  

LAB HEAD: Daniel C. Liebler, Ph.D.

PROVIDER: PXD002042 | Pride | 2015-06-15

REPOSITORIES: Pride

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TCGA-AA-3554-01A-22_W_VU_20121122_A0218_6G_R_FR01.mzML.gz Mzml
TCGA-AA-3554-01A-22_W_VU_20121122_A0218_6G_R_FR01.mzid.gz Mzid
TCGA-AA-3554-01A-22_W_VU_20121122_A0218_6G_R_FR01.pride.mgf.gz Mgf
TCGA-AA-3554-01A-22_W_VU_20121122_A0218_6G_R_FR01.pride.mztab.gz Mztab
TCGA-AA-3554-01A-22_W_VU_20121122_A0218_6G_R_FR01.raw Raw
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Publications

Manipulation of microglial activation as a therapeutic strategy in Alzheimer's disease.

Shie Feng-Shiun FS   Woltjer Randall L RL  

Current medicinal chemistry 20070101 27

Alzheimer's disease (AD) is the leading cause of dementia. Although the etiology of AD remains controversial, the amyloid hypothesis suggests that beta-amyloid (Abeta) peptides may contribute to brain dysfunction, and microglial activation has become increasingly regarded as a potential contributor to disease pathogenesis. Microglial activation is characterized by morphological changes and by production of various effectors, and activated neuroinflammation concurrent with increased oxidative str  ...[more]

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