Ontology highlight
ABSTRACT:
INSTRUMENT(S): LTQ Orbitrap Elite
ORGANISM(S): Homo Sapiens (human)
TISSUE(S): Blood Cell, Primary Cell
DISEASE(S): Disease Free
SUBMITTER: Sharon Pitteri
LAB HEAD: Sharon Pitteri
PROVIDER: PXD007859 | Pride | 2018-01-11
REPOSITORIES: Pride
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2016_UntargetedProteomics.csv | Csv | |||
Key.xlsx | Xlsx | |||
TMT10Plex_1.raw | Raw | |||
TMT10Plex_2.raw | Raw | |||
TMT10Plex_3.raw | Raw |
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Piening Brian D BD Zhou Wenyu W Contrepois Kévin K Röst Hannes H Gu Urban Gucci Jijuan GJ Mishra Tejaswini T Hanson Blake M BM Bautista Eddy J EJ Leopold Shana S Yeh Christine Y CY Spakowicz Daniel D Banerjee Imon I Chen Cynthia C Kukurba Kimberly K Perelman Dalia D Craig Colleen C Colbert Elizabeth E Salins Denis D Rego Shannon S Lee Sunjae S Zhang Cheng C Wheeler Jessica J Sailani M Reza MR Liang Liang L Abbott Charles C Gerstein Mark M Mardinoglu Adil A Smith Ulf U Rubin Daniel L DL Pitteri Sharon S Sodergren Erica E McLaughlin Tracey L TL Weinstock George M GM Snyder Michael P MP
Cell systems 20180117 2
Advances in omics technologies now allow an unprecedented level of phenotyping for human diseases, including obesity, in which individual responses to excess weight are heterogeneous and unpredictable. To aid the development of better understanding of these phenotypes, we performed a controlled longitudinal weight perturbation study combining multiple omics strategies (genomics, transcriptomics, multiple proteomics assays, metabolomics, and microbiomics) during periods of weight gain and loss in ...[more]