Browse
Submit Data
Databases
API
Help

Dataset Information

0 Views

0 Connections

0 Citations

0 Reanalyses

0 Downloads

Omics score: 0

ABSTRACT: rna-seq for Ancherythroculter nigrocauda heart and brain tissues under hypoxia stress

PROVIDER: PRJNA1027844 | ENA |

REPOSITORIES: ENA

ACCESS DATA

Json Xml

Dataset's files

Source:

			Action	DRS
	SRR26379100_1.fastq.gz	Fastqsanger.gz
	SRR26379100_2.fastq.gz	Fastqsanger.gz
	SRR26379101_1.fastq.gz	Fastqsanger.gz
	SRR26379101_2.fastq.gz	Fastqsanger.gz
	SRR26379102_1.fastq.gz	Fastqsanger.gz

Items per page:

1 - 5 of 84

Similar Datasets

Ancherythroculter nigrocauda

Project description:Ancherythroculter nigrocauda overview

| PRJNA207896 | ENA

Ancherythroculter nigrocauda isolate:NO9-14

Project description:Ancherythroculter nigrocauda PacBio WGS

| PRJNA493864 | ENA

Project description:rna-seq for liver and brain tissue of female and male Ancherythroculter nigrocauda

| PRJNA762193 | ENA

Ancherythroculter nigrocauda

Project description:Ancherythroculter nigrocauda Genome sequencing and assembly

| PRJNA533477 | ENA

Ancherythroculter nigrocauda

Project description:Ancherythroculter nigrocauda Genome sequencing and assembly

| PRJNA448477 | ENA

Project description:DNA methylation difference in the liver of Ancherythroculter nigrocauda

| PRJNA890278 | ENA

Project description:Transcriptome analysis of saline-alkali stress on the hybrid (Erythroculter ilishaeformis and Ancherythroculter nigrocauda)

| PRJNA1097380 | ENA

The mouse aging proteomic atlas

Project description:This database provides TMT-labeled proteomic data of aorta (thoracic aorta), brain, heart, kidney, liver, lung, muscle (gastrocnemius muscle), and skin (abdominal skin) of 6, 15, 24, and 30 months old male C57BL/6 mice. In addition to the whole-tissue lysate, low-soluble protein-enriched fraction was also analyzed for heart, kidney, lung, muscle, and skin. Bulk RNA-Seq data are available for brain, heart, kidney, liver, lung, muscle, and skin. The tissues used for transcriptomic analysis and proteomic analysis of whole-tissue lysate and low-soluble protein-enriched fraction were collected from the same mice. All analyses were conducted with 4 biological replicates.

2024-09-17 | PXD040722 | Pride

RNA-seq analysis of Mus caroli and Mus pahari

Project description:We performed RNA-seq experiments Mus caroli and Mus pahari to aid in annotating their genomes. RNA was extracted from multiple tissues: brain, liver, heart and kidney. Complementary ChIP-seq data in this study have also been deposited in ArrayExpress, under accesison number E-MTAB-5769 ( https://www.ebi.ac.uk/arrayexpress/experiments/E-MTAB-5769/ )

2018-03-06 | E-MTAB-5768 | biostudies-arrayexpress

Hypoxic stabilization of RIPOR3 mRNA via METTL3-mediated m6A RNA methylation drives breast tumor growth and metastasis [m6A-seq-sgControl and sgMETTL3]

Project description:N6-methyladenosine (m6A) modification is the most abundant internal mRNA modification in eukaryotes. Hypoxia induces reprogramming of m6A epitranscriptome, but the detail underlying regulator mechanism remains elusive in breast cancer. Here we reported that hypoxia induced elevated m6A modification involved in tumor progression. m6A- sequencing (m6A-seq) combined with RNA sequencing (RNA-seq) identified RIPOR3 as a key target gene of m6A modification under hypoxic stress. Hypoxia-induced increased of RIPOR3 m6A levels promoted its mRNA stability and expression, thereby facilitating the progression and metastasis of breast cancer. Besides, RIPOR3 was overexpressed in breast cancer cells and breast tumor tissues, and elevation of RIPOR3 expression was associated with poor prognosis. Mechanistically, RIPOR3 bound to EGFR and the interaction was enhanced under hypoxia, promoting the activation of the EGFR downstream PI3K-AKT signaling pathway. Altogether, our studies reveal that RIPOR3 responds to hypoxic stress to promote EGFR-PI3K-AKT signal pathway, facilitating breast cancer progression and metastasis, thus presenting itself as a potential therapeutic target.

2024-10-04 | GSE264247 | GEO

OmicsDI is part of the ELIXIR infrastructure

OmicsDI is an Elixir interoperability service. Learn more ›

Tweets

OmicsDI Databases

PRIDE
PeptideAtlas
MassIVE
JPOST Repository
Physiome Model Repository

EGA
EVA
ENA
LINCS
PAXDB
Cell Collective

MetaboLights
Metabolomics Workbench
MetabolomeExpress
GNPS
BioModels
FAIRDOMHub

ArrayExpress
dbGaP
ExpressionAtlas
GEO
NODE

Information

Databases
Help
API
Contact us
Code on GitHub
Terms of use
Submit Data