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Omics score: 0

Mus musculus

ABSTRACT: Liver gene expression from chromogranin A knockout mice (Mahapatra et al. 2005) vs. wild-type mice

OTHER RELATED OMICS DATASETS IN: E-GEOD-18304E-GEOD-18305

PROVIDER: PRJNA123319 | ENA |

REPOSITORIES: ENA

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Gene expression from chromogranin A knockout mice vs. wild-type mice

Project description:This SuperSeries is composed of the following subset Series: GSE18304: Adrenal gland gene expression from chromogranin A knockout mice (Mahapatra et al. 2005) vs. wild-type mice GSE18305: Liver gene expression from chromogranin A knockout mice (Mahapatra et al. 2005) vs. wild-type mice Refer to individual Series

2010-03-03 | E-GEOD-18332 | biostudies-arrayexpress

Mus musculus

Project description:Adrenal gland gene expression from chromogranin A knockout mice (Mahapatra et al. 2005) vs. wild-type mice

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Project description:Liver gene expression from chromogranin A knockout mice (Mahapatra et al. 2005) vs. wild-type mice

2014-07-03 | E-GEOD-18305 | ExpressionAtlas

Liver gene expression from chromogranin A knockout mice (Mahapatra et al. 2005) vs. wild-type mice

Project description:The objective of the experiment is to determine the genes differentially expressed in the liver of the chromogranin A knockout mouse (Mahapatra et al., 2005). Liver from chromogranin A knockout mice (n=3) and wild-type control mice (n=3).

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Liver gene expression profiling of lipoprotein lipase heterozygous knockout mice

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Project description:Cytosolic malic enzyme (Me1) was predicted as a causal gene for abdominal using a novel statistical method named LCMS (Schadt et al., 2005, Nature Genetics). In order to validate this prediction, we profiled the liver tissues of Me1 knockout mice (Me1-/-) and their littermate wild-type (wt) controls to examine the gene expression signature as well as pathways/networks resulting from the single gene perturbation.

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Liver gene expression profiling of complement component 3a receptor 1 knockout (C3ar1-/-) mice

Project description:Complement component 3a receptor 1 (C3ar1) was predicted as a causal gene for abdominal using a novel statistical method named LCMS (Schadt et al., 2005, Nature Genetics). In order to validate this prediction, we profiled the liver tissues of complement component 3a receptor 1 knockout (C3ar1-/-) mice and their littermate wild-type (wt) controls to examine the gene expression signature as well as pathways/networks resulting from the single gene perturbation.

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Project description:Transforming growth factor beta receptor 2 (Tgfbr2) was predicted as a causal gene for abdominal using a novel statistical method named LCMS (Schadt et al., 2005, Nature Genetics). In order to validate this prediction, we profiled the liver tissues of Tgfbr2 heterozygous knockout mice (Tgfbr2+/-) and their littermate wild-type (wt) controls to examine the gene expression signature as well as pathways/networks resulting from the single gene perturbation.

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Liver gene expression from chromogranin A knockout mice (Mahapatra et al. 2005) vs. wild-type mice

Project description:The objective of the experiment is to determine the genes differentially expressed in the liver of the chromogranin A knockout mouse (Mahapatra et al., 2005).

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Project description:Aquaporin-1 Knockout vs Wild type Mice

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