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ABSTRACT: Bacterial profile of Penicillium citrinum SW 171 (PCE-W) extract treated (administered via water) shrimp rearing water after one month (post challenge Vibrio harveyi)

PROVIDER: PRJNA683051 | ENA |

REPOSITORIES: ENA

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			Action	DRS
	SRR13227042.fastq.gz	Fastqsanger.gz
	SRR13227042_1.fastq.gz	Fastqsanger.gz
	SRR13227042_2.fastq.gz	Fastqsanger.gz

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Project description:Bacterial profile of Penicillium citrinum SW 171 (PCE-F) extract treated (administered via feed) shrimp rearing water after one month (post challenge Vibrio harveyi)

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Project description:Bacterial profile of Aspergillus terreus SW155 (ATE-W) extract treated (administered via water) shrimp rearing water after one month (post challenge Vibrio harveyi)

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Project description:Bacterial profile of Aspergillus terreus SW155 (ATE-F) extract treated (administered via feed) shrimp rearing water after one month (post challenge Vibrio harveyi)

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Project description:Bacterial profile of shrimp rearing water after one month (Control- post challenge Vibrio harveyi)

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Project description:Bacterial profile of actinomycete (A3) treated shrimp rearing water after one month (post challenge Vibrio harveyi)

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Identification and expression of salinity-regulated genes in marbled eel (Anguilla marmorata) based on transcriptome analysis

Project description:we used next-generation sequencing technology to characterise mRNA-seq of brackish water (BW, 10‰), fresh water (FW, 0‰), and sea water (SW, 25‰)-treated Anguilla marmorata's gill, kidney and intestine to elucidate the molecular mechanisms of salinity adaptation.

2017-12-10 | GSE95803 | GEO

Project description:Bacterial profile of shrimp rearing water after one month (control for administration of fungal extracts via feed - post challenge Vibrio harveyi)

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Transcriptome data of Penicillium citrinum regulated by pH

Project description:Penicillium citrinum X9-4, which was isolated from infected grapes by our laboratory, produced the highest amount of OTA at pH 5 in culture media, and toxin-production was restrained under acidic environment (pH 3). It revealed the possible mechanism of OTA biosynthesis and metabolic regulation in P. citrinum by transcriptomics, and investigated the reason of OTA biosynthesis was restrained in P. citrinum when cultured under acidic environment.

2017-08-15 | GSE102600 | GEO

Comparative dose-response analysis of liver and kidney transcriptomic effects of trichloroethylene and tetrachloroethylene in B6C3F1 mouse

Project description:Purpose: Trichloroethylene (TCE) and tetrachloroethylene (PCE) are ubiquitous environmental contaminants and occupational health hazards. The goals of this study were to establish the common and differing transcriptional effects of TCE and PCE. This study examined liver and kidney effects of TCE and PCE in a dose-response study design. Methods: Equi-molar doses of TCE (24, 80, 240, 800 mg/kg) or PE (30, 100, 300, 1,000 mg/kg) were administered by gavage in aqueous vehicle to male B6C3F1/J mice. Tissues were collected 24 hrs after exposure. Trichloroacetic acid (TCA), a major oxidative metabolite of both compounds, was measured and RNA sequencing was performed on liver and kidney samples, with ~30 samples for each organ (29 after QC). Results: Most dose-responsive pathways were common among chemicals/tissues, with the strongest effect on peroxisomal beta-oxidation. Effects on liver and kidney mitochondria-related pathways were notably unique to PCE. Tissue-specific acute transcriptional effects of TCE and PCE occurred at human equivalent doses comparable to those for apical effects. Conclusions: Our study is the first RNA-Seq transcriptional study of TCE vs. PCE in both liver and kidney, enabling a detailed comparison of the chemicals and effects on different organs. Our results show strong commonalities of effects, although PCE shows stronger transcriptional responses than TCE for the same equimolar doses.

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Expression data from the cerebral cortex of sugarcane top ethanolic extract (STEE)-administered SAMP8 mice, water-administered SAMP8 mice, and water-administered SAMR1 mice.

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