Browse
Submit Data
Databases
API
Help

Dataset Information

0 Views

0 Connections

0 Citations

0 Reanalyses

0 Downloads

Omics score: 0

Sugar activates defense signaling in Arabidopsis

ABSTRACT: Sugar activates defense signaling in Arabidopsis

PROVIDER: PRJNA716659 | ENA |

REPOSITORIES: ENA

ACCESS DATA

Json Xml

Dataset's files

Source:

			Action	DRS
	SRR14050200.fastq.gz	Fastqsanger.gz
	SRR14050201.fastq.gz	Fastqsanger.gz
	SRR14050202.fastq.gz	Fastqsanger.gz
	SRR14050203.fastq.gz	Fastqsanger.gz
	SRR14050204.fastq.gz	Fastqsanger.gz

Items per page:

1 - 5 of 36

Similar Datasets

Sugar activates defense signaling in Arabidopsis

Project description:Sugar is propose to be a signal molecules for defense in plants. Here, using RNA-seq, we revealed that the application of non-metabolizable glucose analogue 2-deoxy-d-glucose induces the expression of defense-related genes in Arabidopsis. In addition, loss of sugar transporters (STPs) leads to altered responsiveness to flg22, suggesting that sugar-mediated signaling contributes to plant defense.

2024-01-31 | GSE169473 | GEO

Oleic acid-dependent modulation of Nitric Oxide Associated 1 protein levels regulate nitric oxide mediate signaling in plant defense.

2012-08-01 | GSE36797 | GEO

Oleic acid-dependent modulation of Nitric Oxide Associated 1 protein levels regulate nitric oxide mediate signaling in plant defense.

Project description:Oleic acid-dependent modulation of Nitric Oxide Associated 1 protein levels regulate nitric oxide- mediate signaling in plant defense. Nitric Oxide, Oleic acid, Salicylic acid, Arabidopsis, Defense Wild-type Col-0, mutant genotypes ssi2, ssi2 act1 and ssi2 sid2 with three biological replicates were analyzed (one array each)

2012-07-31 | E-GEOD-36797 | biostudies-arrayexpress

An apoplastic peptide activates salicylic acid signaling in Zea mays

Project description:Papain-like cysteine proteases (PLCPs) play important roles in plant defense mechanisms. Previous work identified a set of five apoplastic PLCPs (CP1A, CP1B, CP2, XCP2 and CatB) which are crucial for the orchestration of SA-dependent defense signaling and vice versa in maize (Zea mays). One central question from these findings is which mechanism is triggered by apoplastic PLCPs to induce SA-dependent defenses. By a mass spectrometry approach we discovered a novel peptide (Zip1 = Zea mays immune signaling peptide) to be enriched in apoplastic fluid upon SA treatment. Zip1 induces PR-gene expression when applied to naїve maize leaves. Moreover, it activates apoplastic PLCPs similar as SA does, suggesting Zip1 to play an important role in SA-mediated defense signaling. In vitro studies using recombinant protein showed that CP1A and CP2, but not XCP2 and CatB, release Zip1 from its pro-peptide (PROZIP1) in vitro. Strikingly, metabolite analysis showed direct induction of SA de novo synthesis by Zip1 in maize leaves. In line with this, RNA sequencing revealed that Zip1-mediated changes in maize gene expression largely resemble SA-induced responses. Consequently, Zip1 increases maize susceptibility to the necrotrophic fungal pathogen Botrytis cinerea. In summary, this study identifies the PLCP-released peptide signal Zip1, which triggers SA signaling in maize.

2019-02-15 | PXD006751 | Pride

TMK-based cell surface auxin signaling activates cell wall acidification in Arabidopsis

Project description:The phytohormone auxin controls a myriad of processes in plants, at least in part through its regulation of cell expansion. The "acid growth hypothesis" has been proposed to explain auxin-stimulated cell expansion for five decades, but the mechanism underlying auxin-induced cell wall acidification is poorly characterized. Auxin induces the phosphorylation and activation of the plasma membrane (PM) H+-ATPase that pumps protons into the apoplast, yet how auxin activates its phosphorylation remains elusive. Here, we show that the transmembrane kinase (TMK) auxin signaling proteins interact with PM H+-ATPases and activate their phosphorylation to promote cell wall acidification and hypocotyl cell elongation in Arabidopsis. Auxin induced TMK's interaction with H+-ATPase on the plasma membrane within 1-2 minutes as well as TMK-dependent phosphorylation of the penultimate Thr residue. Genetic, biochemical, and molecular evidence demonstrates that TMKs directly phosphorylate PM H+-ATPase and are required for auxin-induced PM H+-ATPase activation, apoplastic acidification, and cell expansion. Thus, our findings reveal a crucial connection between auxin and PM H+-ATPase activation in regulating apoplastic pH changes and cell expansion via TMK-based cell surface auxin signaling.

2021-07-14 | MSV000087822 | MassIVE

SARS-CoV-2 infection activates AhR signaling

Project description:SARS-CoV-2 infection activates AhR signaling

| PRJNA871921 | ENA

Transcriptome changes triggered by the synthetic defense elicitors DCA and INA in Arabidopsis thaliana

Project description:DCA (3,5-Dichloroanthranilic acid) is a newly identified synthetic defense elicitor. To perform a comparative analysis of defense responses triggered by DCA and the structurally related defense inducer INA (2,6-Dichloroisonicotinic acid) Affymetrix chip experiments were performed with Arabidopsis thaliana seedlings treated with one of these two compounds.

2009-03-20 | GSE13833 | GEO

Transcriptome changes triggered by the synthetic defense elicitors DPMP in Arabidopsis thaliana

Project description:DPMP (2,4-dichloro-6-{(E)-[(3-methoxyphenyl)imino]methyl}phenol ) is a newly identified synthetic defense elicitor. In order to uncover transcriptional patterns associated with defense activation, we profiled by mRNA-seq responses triggered in 14 d-old plate-grown Arabidopsis seedlings by continuous exposure to 3 µM DPMP or Mock.

2016-11-22 | GSE75288 | GEO

RNA-seq-based analysis of the pathogen-induced defense transcriptome in Arabidopsis

Project description:Pathogen infection triggers transcriptional reprogramming in host plants, however we still know little about the dynamics of the pathogen-induced defense transcriptome. The goal of this study was to investigate the dynamic reprogramming of the defense transcriptome in response to Fusarium oxysporum infection in Arabidopsis using RNA-seq technology and to provide a comprehensive analysis of genes underlying the innate immune response against the fungal pathogen. Our results suggest that the Arabidopsis transcriptome is reprogrammed to co-ordinately express multiple positive and negative regulators following pathogen infection to modulate defense gene expression and disease resistance. Our study identified a number of novel genes responsive to pathogen infection and provided a rich source of pathogen responsive genes for further functional characterization.

2012-06-01 | GSE34241 | GEO

Semaphorin activates Hippo signaling to control cell growth

Project description:Semaphorin activates Hippo signaling to control cell growth

| PRJNA580522 | ENA